552410
5-氨基-4-咪唑甲酰胺
95%
别名:
4-氨基-5-氨甲酰咪唑, 4-氨基咪唑-5-甲酰胺, 4-氨基羰基-5-氨基咪唑, 4-甲酰胺基-5-氨基咪唑, 5-氨基-1H-咪唑-4-甲酰胺, 5-氨基-3H-咪唑-4-甲酰胺
登录 查看组织和合同定价。
选择尺寸
关于此项目
经验公式（希尔记法）:
C4H6N4O
化学文摘社编号:
分子量:
126.12
NACRES:
NA.22
PubChem Substance ID:
UNSPSC Code:
12352005
EC Number:
206-641-4
MDL number:
Assay:
95%
Form:
solid
产品名称
5-氨基-4-咪唑甲酰胺, 95%
InChI key
DVNYTAVYBRSTGK-UHFFFAOYSA-N
InChI
1S/C4H6N4O/c5-3-2(4(6)9)7-1-8-3/h1H,5H2,(H2,6,9)(H,7,8)
SMILES string
NC(=O)c1nc[nH]c1N
assay
95%
form
solid
mp
164-170 °C (lit.)
functional group
amide
Quality Level
正在寻找类似产品? 访问 产品对比指南
Application
5-氨基-4-咪唑甲酰胺可用于合成 4-（N'-苯甲酰氨基甲酰）氨基-5-咪唑甲酰胺 和 5-氨基-1- β- D -核糖基-4-咪唑甲酰胺-5'-磷酸盐 (AICAR)。
signalword
Warning
hcodes
Hazard Classifications
Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3
target_organs
Respiratory system
存储类别
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
dust mask type N95 (US), Eyeshields, Gloves
James P White et al.
American journal of physiology. Endocrinology and metabolism, 304(10), E1042-E1052 (2013-03-28)
Although catabolic signaling has a well-established role in muscle wasting during cancer cachexia, the suppression of anabolic signaling also warrants further investigation. In cachectic tumor-bearing mice, circulating IL-6 levels are associated with suppressed muscle protein synthesis and mTORC1 signaling. We
Synthesis of guanosine and its derivatives from 5-amino-l-?-D-ribofuranosyl-4-imidazolecarboxamide. III. Formation of a novel cycloimidazole nucleoside and its cleavage reactions.
Okutsu M and Yamazaki A.
Nucleic Acids Research, 3(1), 237-250 (1976)
Preparation of 5-Amino-1-?-ribosyl-4-imidazolecarboxamide-5'-phosphate and N-(5-Amino-1-?-D-ribosyl-4-imidazolecarbonyl)-L-aspartic Acid 5'-Phosphate.
Huang HT.
Biochemistry, 4(1), 58-62 (1965)
Hiroyasu Hatakeyama et al.
Molecular biology of the cell, 24(6), 809-817 (2013-01-18)
Tbc1d1 is key to skeletal muscle GLUT4 regulation. By using GLUT4 nanometry combined with a cell-based reconstitution model, we uncover a shift in the regulatory mode of Tbc1d1 by showing that Tbc1d1 temporally acquires insulin responsiveness, which triggers GLUT4 trafficking
Takashi Sasaki et al.
American journal of physiology. Endocrinology and metabolism, 306(9), E1085-E1092 (2014-03-20)
Exercise can effectively ameliorate type 2 diabetes and insulin resistance. Here we show that the mRNA levels of one of peroxisome proliferator-activated receptor (PPAR) family members, PPARγ1, and genes related to energy metabolism, including PPARγ coactivator-1 protein-1α (PGC-1α) and lipoprotein
我们的科学家团队拥有各种研究领域经验，包括生命科学、材料科学、化学合成、色谱、分析及许多其他领域.
联系客户支持