表单
liquid
质量水平
浓度
5 M in THF
折射率
n20/D 1.389
密度
0.868 g/mL at 20 °C
0.843 g/mL at 25 °C
官能团
aldehyde
储存温度
2-8°C
SMILES字符串
[H]C(C)=O
InChI
1S/C2H4O/c1-2-3/h2H,1H3
InChI key
IKHGUXGNUITLKF-UHFFFAOYSA-N
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警示用语：
Danger
危险分类
Carc. 1B - Eye Irrit. 2 - Flam. Liq. 2 - Muta. 2 - STOT SE 3
靶器官
Central nervous system, Respiratory system
补充剂危害
储存分类代码
3 - Flammable liquids
WGK
WGK 3
闪点(°F)
-25.1 °F - closed cup
闪点(°C)
-31.7 °C - closed cup
法规信息
新产品
此项目有
Nikoline Borgermann et al.
The EMBO journal, 38(8) (2019-03-28)
DNA-protein crosslinks (DPCs) are highly cytotoxic lesions that obstruct essential DNA transactions and whose resolution is critical for cell and organismal fitness. However, the mechanisms by which cells respond to and overcome DPCs remain incompletely understood. Recent studies unveiled a
Tetsuji Yokoyama et al.
Alcoholism, clinical and experimental research, 29(4), 622-630 (2005-04-19)
Elevated mean corpuscular volume (MCV) is a traditional biological marker for alcohol abuse and alcoholism, but the underlying mechanism is unclear. Three recent epidemiologic studies consistently showed that MCV was elevated by alcohol drinking more markedly among individuals with genetically
Tommaso Mello et al.
Molecular aspects of medicine, 29(1-2), 17-21 (2008-01-01)
Alcohol abuse is one of the major causes of liver fibrosis worldwide. Although the pathogenesis of liver fibrosis is a very complex phenomenon involving different molecular and biological mechanisms, several lines of evidence established that the first ethanol metabolite, acetaldehyde
Hyo-Jung Kwon et al.
Hepatology (Baltimore, Md.), 60(1), 146-157 (2014-02-05)
Aldehyde dehydrogenase 2 (ALDH2) is the major enzyme that metabolizes acetaldehyde produced from alcohol metabolism. Approximately 40-50% of East Asians carry an inactive ALDH2 gene and exhibit acetaldehyde accumulation after alcohol consumption. However, the role of ALDH2 deficiency in the
Mikko Salaspuro
Journal of digestive diseases, 12(2), 51-59 (2011-03-16)
Aldehyde dehydrogenase (ALDH2) and alcohol dehydrogenase (ADH) gene polymorphisms associating with enhanced acetaldehyde exposure and markedly increased cancer risk in alcohol drinkers provide undisputable evidence for acetaldehyde being a local carcinogen not only in esophageal but also in gastric cancer.
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