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Merck
CN

21627-M

Millipore

IHC Select Proteinase K

别名:

Antigen/ Epitope Retrieval Reagent

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关于此项目

UNSPSC代码:
41116010
eCl@ss:
42029053
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包装

pkg of 15 mL

质量水平

制造商/商品名称

Chemicon®
IHC Select

技术

immunohistochemistry: suitable (paraffin)

应用

For use as a Pretreatment Reagent in Immunohistochemical staining procedures. Use Proteinase K to retrieve Epitopes in formalin-fixed, paraffin-embedded tissue sections mounted on glass microscope slides. This procedure is performed after deparaffinization and prior to IHC.



Warm to room temperature before use. Digestion time and dilution should be determined by user as concentration is dependent on the type of antibody, antigen, and fixation method used. Recommended starting dilution range: Neat to 1:30.



For best results use this reagent in conjunction with other IHC Select detection reagents. A complete list of reagents is available at www.chemicon.com.

外形

Liquid Proteinase K containing 15mM sodium azide as a preservative. Discard if it becomes cloudy or contaminated.

制备说明

When stored at 2-8°C, the Proteinase K is stable up to the expiration date printed on the label. Do not freeze or expose to elevated temperatures.

法律信息

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

免责声明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

储存分类代码

12 - Non Combustible Liquids

WGK

nwg

闪点(°F)

Not applicable

闪点(°C)

Not applicable


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Aisan Farhadi et al.
The European journal of neuroscience, 56(1), 3755-3778 (2022-05-06)
Diabetes mellitus may cause tau protein hyperphosphorylation and neurodegeneration, but the exact mechanism by which diabetic conditions induce tau pathology remains unclear. Tau protein hyperphosphorylation is considered a major pathological hallmark of neurodegeneration and can be triggered by diabetes. Various

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