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Merck
CN

AB9708

Anti-DLG4 Antibody

CHEMICON®, rabbit polyclonal

别名:

Post synaptic density 95

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关于此项目

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41
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产品名称

Anti-PSD-95 Antibody, Chemicon®, from rabbit

biological source

rabbit

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

purified by

affinity chromatography

species reactivity

rat

manufacturer/tradename

Chemicon®

technique(s)

western blot: suitable

NCBI accession no.

UniProt accession no.

shipped in

wet ice

target post-translational modification

unmodified

Quality Level

Gene Information

rat ... Dlg4(29495)

Application

Anti-PSD-95 Antibody detects level of PSD-95 & has been published & validated for use in WB.
Research Category
Neuroscience
Research Sub Category
Synapse & Synaptic Biology
Western blot: 0.1-1 μg/mL on rat brain microsomal preparation.

Optimal working dilutions must be determined by the end user.

Biochem/physiol Actions

PSD-95 (Post synaptic density 95). By Western blot the antibody recognizes a band at ~95 kDa in rat brain microsomal preparation.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Immunogen

Synthetic peptide.

Other Notes

Replaces: 04-1066

Physical form

ImmunoAffinity Purified
Liquid in PBS with 0.1% sodium azide.

Preparation Note

Maintain at 2-8°C in undiluted aliquots for up to 6 months after date of receipt.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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存储类别

12 - Non Combustible Liquids

wgk

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable


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Sergei V Baranov et al.
Proceedings of the National Academy of Sciences of the United States of America, 116(2), 650-659 (2018-12-26)
Neuritic retraction in the absence of overt neuronal death is a shared feature of normal aging and neurodegenerative disorders, but the intracellular mechanisms modulating this process are not understood. We propose that cumulative distal mitochondrial protein damage results in impaired
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Although the molecular mechanism is not clear, the clinically tested drug ketamine has rapid antidepressant action that does not require the multiple weeks of treatment needed for other antidepressant drugs to have an effect. We showed that ketamine potentiated Schaffer
Ram Kuwar et al.
Journal of Alzheimer's disease : JAD, 82(4), 1769-1783 (2021-07-06)
Alzheimer's disease (AD) is a progressive neurodegenerative disorder, and the most common type of dementia. A growing body of evidence has implicated neuroinflammation as an essential player in the etiology of AD. Inflammasomes are intracellular multiprotein complexes and essential components
Marie Pronot et al.
Frontiers in molecular neuroscience, 14, 780535-780535 (2021-12-11)
Synapses are highly specialized structures that interconnect neurons to form functional networks dedicated to neuronal communication. During brain development, synapses undergo activity-dependent rearrangements leading to both structural and functional changes. Many molecular processes are involved in this regulation, including post-translational
Marta U Wołoszynowska-Fraser et al.
eNeuro, 8(5) (2021-08-22)
Retinoic acid (RA), a metabolite of vitamin A, has many physiological functions, and mounting evidence points to important roles in cognition. In vitro experiments indicate that RA is involved in homeostatic synaptic scaling in the hippocampus, which supports overall network

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