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Merck
CN

249882

三氯化铝 溶液

1.0 M in nitrobenzene

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关于此项目

经验公式(希尔记法):
AlCl3
化学文摘社编号:
分子量:
133.34
MDL编号:
UNSPSC代码:
12352302
PubChem化学物质编号:
NACRES:
NA.55
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表单

liquid

反应适用性

reagent type: catalyst
core: aluminum

浓度

0.95-1.10 M (EDTA titration)
1.0 M in nitrobenzene

密度

1.249 g/mL at 25 °C

SMILES字符串

Cl[Al](Cl)Cl

InChI

1S/Al.3ClH/h;3*1H/q+3;;;/p-3

InChI key

VSCWAEJMTAWNJL-UHFFFAOYSA-K

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警示用语:

Danger

危险分类

Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Chronic 3 - Carc. 2 - Eye Dam. 1 - Repr. 1B - Skin Corr. 1B - STOT RE 1 Inhalation

靶器官

Blood

补充剂危害

储存分类代码

6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials

WGK

WGK 3

闪点(°F)

190.4 °F - closed cup

闪点(°C)

88 °C - closed cup

法规信息

危险化学品
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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Acceleration of the Diels-Alder reaction by aluminum chloride.
Yates P and Eaton P.
Journal of the American Chemical Society, 82(16), 4426-4437 (1960)
Zhiliang Huang et al.
Organic & biomolecular chemistry, 11(11), 1810-1814 (2013-02-08)
AlCl(3) promoted Friedel-Crafts acylation between 4-tert-butylbenzoyl chloride and mesitylene was investigated. The donor-acceptor complex was observed as the major species. Kinetic investigation demonstrated that the reaction was first-order on the donor-acceptor complex and zero-order on ArH, suggesting that the donor-acceptor
Amination of< i> exo</i>-2-chloronorbornane and norbornane with trichloramine-aluminum chloride.
Kovacic P, et al.
Tetrahedron, 26(2), 529-538 (1970)
Hai-hua Zhao et al.
Behavioural brain research, 241, 228-234 (2012-12-12)
Environmental agent aluminum, a well-known neurotoxin, has been proposed to play a role in the development of Alzheimer's disease (AD), and produced clinical and pathological features which were strikingly similar to those seen in AD brain, such as neurofibrillary tangles.
Dharmalingam Prakash et al.
Neuromolecular medicine, 15(1), 192-208 (2013-01-15)
Aluminum (Al) is an environmental neurotoxin that affects cerebral functions and causes health complications. However, the role of Al in arbitrating glia homeostasis and pathophysiology remains obscure. Astrocyte, microglia activation (reactive gliosis), and associated inflammatory events play a decisive role

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