H3131
2-羟雌甾二醇
≥95% (HPLC)
别名:
1,3,5(10)-雌甾三烯-2,3-17β-三醇, 雌甾-1,3,5(10)-三烯2,3,17-三醇
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关于此项目
经验公式（希尔记法）:
C18H24O3
化学文摘社编号:
分子量:
288.38
NACRES:
NA.77
PubChem Substance ID:
UNSPSC Code:
12352200
MDL number:
biological source
synthetic (organic)
sterility
non-sterile
assay
≥95% (HPLC)
form
powder
solubility
ethanol: 20 mg/mL, clear, faintly yellow to yellow
shipped in
ambient
storage temp.
−20°C
SMILES string
C[C@]12CC[C@H]3[C@@H](CCc4cc(O)c(O)cc34)[C@@H]1CC[C@@H]2O
InChI
1S/C18H24O3/c1-18-7-6-11-12(14(18)4-5-17(18)21)3-2-10-8-15(19)16(20)9-13(10)11/h8-9,11-12,14,17,19-21H,2-7H2,1H3/t11-,12+,14-,17-,18-/m0/s1
InChI key
DILDHNKDVHLEQB-XSSYPUMDSA-N
Gene Information
rat ... Ar(24208)
R K Babbs et al.
Physiology & behavior, 103(5), 508-512 (2011-04-19)
One conundrum of binge eating is that women are more likely to suffer from binge-related disorders, even though estradiol decreases food intake. 2-hydroxyestradiol (2OHE2), an estrogen metabolite, may account for the contradiction, due to possible interference with DA signaling. We
Weidong Geng et al.
Journal of cellular physiology, 220(2), 332-340 (2009-04-11)
High [HCO(3)(-)] inhibits and low [HCO(3)(-)] stimulates bone resorption, which mediates part of the effect of chronic acidosis or acid feeding on bone. Soluble adenylyl cyclase (sAC) is a bicarbonate sensor that can potentially mediate the effect of bicarbonate on
Water-soluble products of the estrogens. Bile metabolites of 2-hydroxyestradiol-17beta in man.
J S Elce et al.
The Journal of clinical endocrinology and metabolism, 36(5), 1027-1030 (1973-05-01)
G Basini et al.
The Journal of endocrinology, 199(1), 127-135 (2008-07-30)
The rapid, controlled, and cyclical nature of angiogenesis in the ovarian follicle suggests the potential for sex steroids to influence neovascularization. Angiogenesis is regulated by a local balance between the levels of endogenous stimulators and inhibitors. Multiple lines of evidence
Tara M D'Eon et al.
Obesity (Silver Spring, Md.), 16(6), 1284-1288 (2008-04-19)
Systemic loss of estradiol (E2) during menopause is associated with increased adiposity which can be prevented with E2 replacement. Rodent studies suggest that E2, or lack of, is a key mediator in menopause-related metabolic changes. We have previously demonstrated that
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