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Merck
CN

I3389

Sigma-Aldrich

D-myo-Inositol 1,3,4-tris-phosphate ammonium salt

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关于此项目

线性分子式:
C6H15O15P3 · 6NH3
化学文摘社编号:
分子量:
522.28
MDL编号:
UNSPSC代码:
12352201
PubChem化学物质编号:
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储存温度

−20°C

SMILES字符串

OC1C(O)C(OP(O)(O)=O)C(OP(O)(O)=O)C(O)C1OP(O)(O)=O

InChI

1S/C6H15O15P3/c7-1-2(8)5(20-23(13,14)15)6(21-24(16,17)18)3(9)4(1)19-22(10,11)12/h1-9H,(H2,10,11,12)(H2,13,14,15)(H2,16,17,18)

InChI key

MMWCIQZXVOZEGG-UHFFFAOYSA-N

生化/生理作用

Occurs naturally in carbachol-stimulated rat parotid glands1 and in adrenal glomerular cells.2

储存分类代码

11 - Combustible Solids

WGK

WGK 3

闪点(°F)

Not applicable

闪点(°C)

Not applicable

法规信息

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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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H Plattner et al.
Cell calcium, 51(5), 351-382 (2012-03-06)
The importance of Ca2+-signaling for many subcellular processes is well established in higher eukaryotes, whereas information about protozoa is restricted. Recent genome analyses have stimulated such work also with Alveolates, such as ciliates (Paramecium, Tetrahymena) and their pathogenic close relatives
Jun Wang et al.
Developmental biology, 302(1), 143-153 (2006-10-10)
Integrin signaling modulates trophoblast adhesion to extracellular matrices during blastocyst implantation. Fibronectin (FN)-binding activity on the apical surface of trophoblast cells is strengthened after elevation of intracellular Ca(2+) downstream of integrin ligation by FN. We report here that phosphoinositide-specific phospholipase
Gilda A Nusco et al.
Biochemical and biophysical research communications, 348(1), 109-114 (2006-08-01)
Cofilin is a small protein that belongs to the family of actin-depolymerizing factors (ADF). The main cellular function of cofilin is to change cytoskeletal dynamics and thus to modulate cell motility and cytokinesis. We have recently demonstrated that the actin
Hao Du et al.
Plant molecular biology, 77(6), 547-563 (2011-11-01)
Drought and salt stresses are major limiting factors for crop production. To identify critical genes for stress resistance in rice (Oryza sativa L.), we screened T-DNA mutants and identified a drought- and salt-hypersensitive mutant dsm3. The mutant phenotype was caused
B Q Phillippy et al.
Free radical biology & medicine, 22(6), 939-946 (1997-01-01)
Iron chelates of inositol 1,2,3-trisphosphate and inositol 1,2,3,6-tetrakisphosphate lacked free coordination sites and prevented the iron-catalyzed oxidation of ascorbic acid and peroxidation of arachidonic acid. In contrast, iron chelates of inositol 1,2,6-trisphosphate and inositol 1,2,5,6-tetrakisphosphate contained available coordination sites, permitted

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