SAB1306488
ANTI-CD166(N-TERMINAL) antibody produced in rabbit
purified immunoglobulin, buffered aqueous solution
别名:
ALCAM, Activated leukocyte cell adhesion molecule, CD166 antigen, CD_antigen=CD166, MEMD
biological source
rabbit
antibody form
purified immunoglobulin
antibody product type
primary antibodies
clone
polyclonal
form
buffered aqueous solution
mol wt
65102 Da
species reactivity
human
species reactivity (predicted by homology)
mouse, bovine, rat, rabbit
technique(s)
ELISA: suitable, flow cytometry: 1:10-1:50, immunohistochemistry: 1:50-1:100, western blot: 1:1000
NCBI accession no.
UniProt accession no.
shipped in
wet ice
storage temp.
−20°C
Gene Information
human ... ALCAM(214)
Biochem/physiol Actions
This CD166 antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 58-87 amino acids from the N-terminal region of human CD166.
Physical form
Supplied in PBS with 0.09% (W/V) sodium azide
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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存储类别
12 - Non Combustible Liquids
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
常规特殊物品
此项目有
Takamune Achiha et al.
PloS one, 15(12), e0243272-e0243272 (2020-12-04)
Cluster of differentiation (CD) 166 or activated leukocyte cell adhesion molecule (ALCAM) is a transmembrane molecule known to be an intercellular adhesion factor. The expression and function of ALCAM in medulloblastoma (MB), a pediatric brain tumor with highly advanced molecular
Y Chu et al.
Cancer gene therapy, 21(5), 200-208 (2014-05-24)
Anti-angiogenesis gene therapy is considered a promising treatment for excessive vascularization. Mesenchymal stem cell (MSC)-based gene therapy may enhance the effect of anti-angiogenesis by maintaining a long therapeutic period in vivo. However, transduction efficiencies and transgene expression in MSC-based gene
Hung-Li Tsai et al.
PloS one, 9(8), e104937-e104937 (2014-08-30)
Wnts were previously shown to regulate the neurogenesis of neural stem or progenitor cells. Here, we explored the underlying molecular mechanisms through which Wnt signaling regulates neurotrophins (NTs) in the NT-induced neuronal differentiation of human mesenchymal stem cells (hMSCs). NTs
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