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Carlos Gómez-Martin et al.
Journal of clinical pathology, 65(8), 751-757 (2012-05-10)
To study the HER2 gene amplification or overexpression in patients with advanced gastric cancer (GC) and their association with patient characteristics and patient survival. Tumour tissue samples from 148 patients with advanced GC were studied for HER2 by immunohistochemistry (IHC)
Mary W Meagher et al.
Brain, behavior, and immunity, 21(8), 1083-1095 (2007-06-27)
Prior exposure to social disruption stress (SDR) exacerbates both the acute and chronic phase of Theiler's murine encephalomyelitis virus infection (TMEV; [Johnson, R.R., Storts, R., Welsh, T.H., Jr., Welsh, C.J., Meagher, M.W., 2004. Social stress alters the severity of acute
Jürgen Knapp et al.
Shock (Augusta, Ga.), 43(6), 576-581 (2015-02-24)
The immunosuppressant drug cyclosporine A (CsA) is a direct inhibitor of the mitochondrial permeability transition pore, which is the common end point of many pathways of ischemic preconditioning and postconditioning. We studied the neuroprotective and cardioprotective effect of CsA after
Matthias Schwab et al.
Clinical pharmacology and therapeutics, 78(6), 627-634 (2005-12-13)
The clinical outcome of acid-related disorders treated by proton pump inhibitors (PPIs) might be dependent on the polymorphically expressed cytochrome P450 (CYP) 2C19, which is involved in PPI metabolism. We tested whether esomeprazole-induced healing of gastroesophageal reflux disease (GERD) is
F Z Stanczyk et al.
American journal of obstetrics and gynecology, 165(6 Pt 1), 1837-1842 (1991-12-01)
To determine whether the adrenal androgen 11 beta-hydroxyandrostenedione is a more sensitive and specific marker than dehydroepiandrosterone sulfate, we compared these serum androgens in 81 women with anovulatory hyperandrogenism before treatment, after corticotropin and corticotropin-releasing-factor stimulation, and after short- and
Kwong-Ming Kee et al.
Digestive diseases and sciences, 58(2), 556-561 (2012-09-25)
The long-term trend of platelet count in chronic hepatitis C virus patients with sustained virological response (SVR) has rarely been investigated. To elucidate changes of thrombocytopenia after SVR, trajectory patterns of platelet count over time and their associated factors. From
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