488-17-5

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Exonucleolytic degradation of high-density labeled DNA studied by fluorescence correlation spectroscopy.

Nicky Ehrlich et al.

The Analyst, 137(5), 1160-1167 (2012-01-24)

The exonucleolytic degradation of high-density labeled DNA by exonuclease III was monitored using two-color fluorescence correlation spectroscopy (FCS). One strand of the double stranded template DNA was labeled on either one or two base types and additionally at one end

Diabetic hypertensive leptin receptor-deficient db/db mice develop cardioregulatory autonomic dysfunction.

Andrey C da Costa Goncalves et al.

Hypertension (Dallas, Tex. : 1979), 53(2), 387-392 (2008-11-26)

Leptin receptor-deficient db/db mice develop human type 2 diabetes mellitus, hypertension, and obesity with disrupted circadian blood pressure (BP) rhythm. Whether leptin is the sole mechanism mediating autonomic imbalance and hypertension is unclear. To explore this notion further, we measured

RhCE protein variants in Southwestern Germany detected by serologic routine testing.

Peter Bugert et al.

Transfusion, 49(9), 1793-1802 (2009-05-21)

Variant RHCE alleles with diminished expression of C, c, E, and e antigens have been described and indicate the genetic diversity of this gene locus in several populations. In this study the molecular background of variant RhCE antigens identified by

Targeted deletion of p53 prevents cardiac rupture after myocardial infarction in mice.

Hidenori Matsusaka et al.

Cardiovascular research, 70(3), 457-465 (2006-03-15)

Apoptosis may play an important role in cardiac remodeling after myocardial infarction (MI). p53 is a well-known proapoptotic factor. However, its pathophysiological significance in these conditions remains unclear. We thus examined the effects of target deletion of the p53 gene

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