InChI key
VZPGINJWPPHRLS-UHFFFAOYSA-N
InChI
1S/C12H10N4/c13-7-5-11-12(6-8(7)14)16-10-4-2-1-3-9(10)15-11/h1-6H,13-14H2
SMILES string
Nc1cc2nc3ccccc3nc2cc1N
assay
90%
form
solid
mp
>300 °C
Quality Level
signalword
Warning
hcodes
Hazard Classifications
Acute Tox. 4 Oral - Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3
target_organs
Respiratory system
存储类别
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
dust mask type N95 (US), Eyeshields, Gloves
G Grosa et al.
Xenobiotica; the fate of foreign compounds in biological systems, 34(4), 345-352 (2004-07-23)
1. The results of an in vitro study of the metabolism of benzofuroxan using either cytosolic or microsomal fractions obtained from rat liver are reported. 2. Benzofuroxan was incubated with an appropriate volume of cytosol or microsomal suspension; control incubations
Patty K-L Fu et al.
Photochemistry and photobiology, 81(1), 89-95 (2004-10-21)
Aromatic amines, such as o-phenylenediamine (OPD), have been used extensively in commercial hair dyes and in the synthesis of agricultural pesticides. Air oxidation of OPD results in the formation of 2,3-diaminophenazine (DAP). Although the mutagenic toxicity of DAP has been
R P Doyle et al.
Acta crystallographica. Section C, Crystal structure communications, 57(Pt 1), 104-105 (2001-02-15)
The planar electron-rich heterocyclic diamine 2,3-diaminophenazine (DAP), C12H10N4, is of particular interest to both chemists and biochemists because of its rich organic chemistry and intense luminescence. In this paper, we report the first structure of DAP in its non-protonated form
P S Oturai et al.
Metabolism: clinical and experimental, 49(8), 996-1000 (2000-08-23)
The accelerated formation of advanced glycation end-products (AGEs) due to elevated glycemia has repeatedly been reported as a central pathogenic factor in the development of diabetic microvascular complications. The effects of a novel inhibitor of AGE formation, NNC39-0028 (2,3-diaminophenazine), and
T Watanabe et al.
Mutation research, 245(1), 15-22 (1990-09-01)
The mutagenicity of o- and m-phenylenediamine (PD) was remarkedly enhanced by oxidation; their major mutagenic oxidation products were 2,3- and 2,7-diaminophenazine, respectively. In order to evaluate the modulation effect of p-PD on the oxidation of m- or o-PD, p-PD and
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