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Merck
CN

14409

Sigma-Aldrich

purum, ≥99%, powder

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关于此项目

经验公式(希尔记法):
Zn
化学文摘社编号:
分子量:
65.39
EC 号:
MDL编号:
UNSPSC代码:
12352300
PubChem化学物质编号:
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蒸汽压

1 mmHg ( 487 °C)

等级

purum

方案

≥99%

表单

powder

电阻率

5.8 μΩ-cm, 20°C

沸点

907 °C (lit.)

mp

420 °C (lit.)

密度

7.133 g/mL at 25 °C (lit.)

痕量阳离子

As: ≤0.2 mg/kg
Cd: ≤50 mg/kg
Fe: ≤500 mg/kg
Pb: ≤500 mg/kg
Sn: ≤100 mg/kg

SMILES字符串

[Zn]

InChI

1S/Zn

InChI key

HCHKCACWOHOZIP-UHFFFAOYSA-N

应用

Reducing agent; used for preparation of organozinc reagents, Reformatsky reagents, and the Simmons-Smith reagent.

象形图

Environment

警示用语:

Warning

危险声明

预防措施声明

危险分类

Aquatic Acute 1 - Aquatic Chronic 1

储存分类代码

13 - Non Combustible Solids

WGK

WGK 2

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

Eyeshields, Gloves

法规信息

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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Marino Petrini et al.
The Journal of organic chemistry, 67(13), 4530-4535 (2002-06-22)
Alpha-amidoalkylphenyl sulfones behave as N-acylimino equivalents in the reaction with functionalized allylzinc reagents. The addition products obtained using the zinc derivative of ethyl 2-(bromomethyl)acrylate can be readily transformed into alpha-methylene-gamma-lactams using different cyclization procedures. The allylzinc reagent obtained from 3-bromo-1-acetoxy-1-propene
Hyacinthe Fillon et al.
Journal of the American Chemical Society, 125(13), 3867-3870 (2003-03-27)
A new chemical method for the preparation of arylzinc intermediates is described in acetonitrile, on the basis of the activation of aryl bromides by low-valent cobalt species arising from the reduction of cobalt halide by zinc dust. This procedure allows
Knochel, P.
Chemical Reviews, 93, 217-217 (1993)
A viewpoint about the treatment of Wilson's disease.
Abdul Qayyum Rana et al.
The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques, 40(4), 612-614 (2013-06-22)
Sara Eyal et al.
Journal of nuclear medicine : official publication, Society of Nuclear Medicine, 50(5), 798-806 (2009-05-01)
Studies in rodents indicate that the disruption of P-glycoprotein (P-gp) function increases drug distribution into the developing fetus and organs such as the brain. To simultaneously and serially evaluate the effect of P-gp activity and inhibition on the tissue distribution

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