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Merck
CN

164453

2-噻吩甲酰胺

99%

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关于此项目

经验公式(希尔记法):
C5H5NOS
化学文摘社编号:
分子量:
127.16
NACRES:
NA.22
PubChem Substance ID:
UNSPSC Code:
12352100
MDL number:
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InChI

1S/C5H5NOS/c6-5(7)4-2-1-3-8-4/h1-3H,(H2,6,7)

SMILES string

NC(=O)c1cccs1

InChI key

DENPQNAWGQXKCU-UHFFFAOYSA-N

assay

99%

mp

181-183 °C (lit.)

存储类别

11 - Combustible Solids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

新产品
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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Hyunji Lee et al.
Journal of Korean medical science, 25(11), 1574-1581 (2010-11-10)
The massive reorganization of microtubule network involves in transcriptional regulation of several genes by controlling transcriptional factor, nuclear factor-kappa B (NF-κB) activity. The exact molecular mechanism by which microtubule rearrangement leads to NF-κB activation largely remains to be identified. However
Yunfei Tan et al.
Pharmacology, biochemistry, and behavior, 188, 172839-172839 (2019-12-24)
The N-methyl-d-aspartate receptor (NMDAR) antagonists including phencyclidine (PCP) and ketamine produce cognitive deficits in rodents and humans. We previously reported that (R)-ketamine produced the beneficial effects compared to (S)-ketamine in several animal models including depression. Here we compared the effects
Q Ren et al.
Translational psychiatry, 5, e666-e666 (2015-10-28)
Depression is a core symptom of methamphetamine (METH) withdrawal during the first several weeks of abstinence. However, the precise mechanisms underlying METH withdrawal symptoms remain unknown. Brain-derived neurotrophic factor (BDNF) and its specific receptor, tropomyosin-related kinase (TrkB), have a role
Tegan Triggs et al.
Expert opinion on investigational drugs, 29(5), 507-523 (2020-04-16)
Preterm birth is the leading cause of neonatal morbidity and mortality globally and poses a substantial economic burden. Consequently, there is a need for the identification of therapeutic targets and novel experimental drugs for the inhibition of preterm labor to
Magen E LaPorte et al.
Theriogenology, 95, 8-17 (2017-05-04)
In previous work, an EP2 prostanoid receptor (EP2R) agonist in vivo increased mRNA expression of luteal LH receptors (LHR), unoccupied and occupied luteal; LHR, and circulating progesterone, while an EP3R or FPR agonist decreased; mRNA expression of luteal LHR, unoccupied and

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