194751
N-叔丁基羟胺盐酸盐
≥98%
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线性分子式:
(CH3)3CNHOH · HCl
化学文摘社编号:
分子量:
125.60
UNSPSC Code:
12352100
NACRES:
NA.22
PubChem Substance ID:
EC Number:
260-771-6
Beilstein/REAXYS Number:
3546053
MDL number:
产品名称
N-叔丁基羟胺盐酸盐, ≥98%
InChI key
DCSATTBHEMKGIP-UHFFFAOYSA-N
InChI
1S/C4H11NO.ClH/c1-4(2,3)5-6;/h5-6H,1-3H3;1H
SMILES string
Cl.CC(C)(C)NO
assay
≥98%
form
solid
mp
183-185 °C (lit.)
Quality Level
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Application
N-tert-Butylhydroxylamine hydrochloride was used in spin trapping of short-lived radicals. It was also used in the synthesis of α-ketoamides and 3-spirocyclopropanated 2-azetidinones.
存储类别
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
Hyun Jeong Kim et al.
Redox report : communications in free radical research, 10(6), 287-293 (2006-01-28)
Heat shock may increase oxidative stress due to increased production of reactive oxygen species and/or the promotion of cellular oxidation events. Therefore, compounds that scavenge reactive oxygen species may regulate heat shock-induced cell death. Recently, it has been shown that
Yu-Kyung Kim et al.
Clinical hemorheology and microcirculation, 40(4), 315-324 (2009-01-08)
Irradiation has been shown to induce biochemical changes in stored red blood cells (RBCs) and to generate reactive oxygen species (ROS). This study evaluated the hemorheological properties, the degree of lipid peroxidation and the oxidative susceptibility of irradiated RBCs. Furthermore
David W Killilea et al.
Annals of the New York Academy of Sciences, 1019, 365-367 (2004-07-13)
Iron accumulates as a function of age and is associated with the pathology of numerous age-related diseases. These changes may be caused by altered iron homeostasis at the cellular level, yet this is poorly understood. Therefore, changes in iron content
David W Killilea et al.
Antioxidants & redox signaling, 5(5), 507-516 (2003-10-29)
Iron accumulates as a function of age in several tissues in vivo and is associated with the pathology of numerous age-related diseases. The molecular basis of this change may be due to a loss of iron homeostasis at the cellular
Synthesis of alpha-ketoamides by a molecular-sieves-promoted formal oxidative coupling of aliphatic aldehydes with isocyanides.
Jean-Marie Grassot et al.
Angewandte Chemie (International ed. in English), 47(5), 947-950 (2007-12-20)
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