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286575

2-Methoxyestradiol

98%

别名:

1,3,5(10)-Estratriene-2,3,17-triol 2-methyl ether, 2,3,17β-Trihydroxy-1,3,5(10)-estratriene 2-methyl ether, 2-Hydroxyestradiol 2-methyl ether, 2-Methoxyestra-1,3,5(10)-triene-3,17β-diol, 3,17β-Dihydroxy-2-methoxy-1,3,5(10)-estratriene

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关于此项目

经验公式（希尔记法）:
C19H26O3
化学文摘社编号:
362-07-2
分子量:
302.41
UNSPSC Code:
51111800
PubChem Substance ID:
329753282
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assay

98%

InChI

1S/C19H26O3/c1-19-8-7-12-13(15(19)5-6-18(19)21)4-3-11-9-16(20)17(22-2)10-14(11)12/h9-10,12-13,15,18,20-21H,3-8H2,1-2H3/t12?,13?,15?,18?,19-/m0/s1

InChI key

CQOQDQWUFQDJMK-VKYAZVNUSA-N

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Danger

Hazard Classifications

Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1B - Eye Irrit. 2 - Repr. 1B - Skin Irrit. 2 - STOT RE 1 - STOT SE 3

target_organs

Respiratory system

存储类别

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges

法规信息

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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number
06054DG
05433MF
05338AS
04452BR
24737DN

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A P Kumar et al.
Molecular carcinogenesis, 31(3), 111-124 (2001-08-02)
2-Methoxyestradiol (2-ME), an endogenous metabolite of 17beta-estradiol, is present in human blood and urine. Here we show for the first time that 2-ME significantly inhibited the growth of normal prostate epithelial cells and androgen-dependent LNCaP and androgen-independent DU145 prostate cancer
Nehal J Lakhani et al.
Rapid communications in mass spectrometry : RCM, 19(9), 1176-1182 (2005-04-09)
A liquid chromatography/tandem mass spectrometric (LC/MS/MS) assay was developed for the quantitative determination of 2-methoxyestradiol (2ME2) in human plasma. Sample pretreatment involved liquid-liquid extraction with ethyl acetate of 0.3-mL aliquots of plasma spiked with the internal standard, deuterated 2ME2 (2ME2-d5).
S Sarkar et al.
Oncogene, 33(1), 26-33 (2012-12-19)
The androgen receptor (AR) has a vital role in the onset and progression of prostate cancer by promoting G1-S progression, possibly by functioning as a licensing factor for DNA replication. We here report that low dose 2-methoxyestradiol (2-ME), an endogenous
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