292265
N,N′-二苯基-对苯二胺
98%
别名:
N,N′-二苯基-1,4-苯二胺
质量水平
方案
98%
沸点
220-225 °C/0.5 mmHg (lit.)
mp
143-145 °C (lit.)
SMILES字符串
N(c1ccccc1)c2ccc(Nc3ccccc3)cc2
InChI
1S/C18H16N2/c1-3-7-15(8-4-1)19-17-11-13-18(14-12-17)20-16-9-5-2-6-10-16/h1-14,19-20H
InChI key
UTGQNNCQYDRXCH-UHFFFAOYSA-N
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应用
- TPA诱导的细胞间隙连接通讯的抑制不受自由基介导：该研究探讨了N,N-二苯基对苯二胺（DPPD）在细胞通讯中的作用，提供了有关其在高性能橡胶稳定剂和生命科学制造化学品中的应用的见解。（Hasler et al., 1990）。
警示用语：
Warning
危险声明
危险分类
Aquatic Chronic 3 - Skin Sens. 1
储存分类代码
11 - Combustible Solids
WGK
WGK 1
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
dust mask type N95 (US), Eyeshields, Faceshields, Gloves
Yoshiko Kawai et al.
Journal of pharmacological sciences, 111(4), 433-439 (2009-12-08)
Cisplatin causes chronic interstitial disease with fibrosis, but the development mechanism of interstitial fibrosis is not yet understood. We examined the effect of an antioxidant, N,N'-diphenyl-1,4-phenylenediamine (DPPD), on development of interstitial fibrosis induced by cisplatin. Cisplatin increased blood urea nitrogen
Sung Min Park et al.
Pharmacology & toxicology, 92(1), 43-50 (2003-04-25)
This study was undertaken in order to examine the roles of lipid peroxidation and poly (ADP-ribose) polymerase (PARP) activation in oxidant-induced renal cell death. Opossum kidney cell cultures were used as the renal epithelial cell model, and an inorganic hydroperoxide
Xiaobo Ji et al.
Analytical sciences : the international journal of the Japan Society for Analytical Chemistry, 23(11), 1317-1320 (2007-11-14)
The oxidation of N,N'-diphenyl-1,4-phenylenediamine (DPPD) has been studied by cyclic voltammetry at glassy carbon electrodes in propylene carbonate. Next, the reaction between ammonia and DPPD was similarly investigated. It has been shown that ammonia reversibly removes protons from oxidized DPPD
A Campos-Neto et al.
Tuberculosis (Edinburgh, Scotland), 81(5-6), 353-358 (2002-01-22)
Although the tuberculin test has aided in the diagnosis of tuberculosis for more than 85 years, its interpretation is difficult particularly because sensitization with non-tuberculous mycobacteria leads to false positive tests. Using the guinea pig model of tuberculosis, we have
J A Kotarek et al.
Analytical biochemistry, 399(1), 30-38 (2009-12-19)
Evidence that membrane-associated amyloid aggregate growth can impart membrane damage represents one possible mechanism for the neurodegeneration associated with deposited amyloid-beta protein (Abeta) aggregates in the brains of Alzheimer's disease (AD) patients. This potential pathogenic event necessitates an understanding of
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