690228
N,N-双[3-(甲氨基)丙基]甲胺
98%
别名:
N-甲基-3,3′-双(甲氨基)二丙胺, N-甲基-N,N-双[3-(甲氨基)丙基]胺, 甲基双(3-甲基氨丙基)胺
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线性分子式:
CH3N(CH2CH2CH2NHCH3)2
化学文摘社编号:
分子量:
173.30
UNSPSC Code:
12352100
NACRES:
NA.22
PubChem Substance ID:
EC Number:
204-645-0
Beilstein/REAXYS Number:
6711228
MDL number:
Assay:
≥97.5% (GC), 97.5-102.5% (with HClO4, T), 98%
Form:
liquid
InChI key
PQYGBJHVVYREGU-UHFFFAOYSA-N
InChI
1S/C9H23N3/c1-10-6-4-8-12(3)9-5-7-11-2/h10-11H,4-9H2,1-3H3
SMILES string
CNCCCN(C)CCCNC
assay
≥97.5% (GC), 97.5-102.5% (with HClO4, T), 98%
form
liquid
impurities
≤1% water
signalword
Danger
hcodes
Hazard Classifications
Skin Corr. 1B
存储类别
8A - Combustible corrosive hazardous materials
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter
法规信息
新产品
此项目有
S Levine et al.
Journal of immunological methods, 54(3), 355-360 (1982-11-12)
In the past, the lesions of experimental allergic encephalomyelitis (EAE) have been induced to localize around brain tissue damaged by anoxia or direct physical or chemical attack. The procedure for producing the requisite antecedent brain injury has been simplified by
Gold thioglucose, mimpa and lesions of the ventromedial hypothalamus.
D F Brown
Journal of neuropathology and experimental neurology, 43(4), 455-457 (1984-07-01)
D Nochlin et al.
Journal of neuropathology and experimental neurology, 41(2), 233-240 (1982-03-01)
The administration of an aliphatic triamine, 3,3'-methylimino-bis-(N-methylpropylamine), was used to produce non-necrotizing lesions in the hypothalamus and medulla oblongata of rats. The distribution of the lesions in the hypothalamus was similar to that produced by goldthioglucose, and consisted ultrastructurally of
S Levine et al.
Brain research, 242(2), 219-225 (1982-06-24)
An aliphatic triamine has been reported to cause lesions in rats in the vicinity of the area postrema and the median eminence of the hypothalamus, sites known to lack a blood-brain barrier. The present study revealed that some of the
J C Lyons et al.
Journal of neuropathology and experimental neurology, 41(1), 45-53 (1982-01-01)
The clinical and pathologic features of two brothers with the adult form of Gaucher's disease, both of whom developed cerebral glioblastoma multiforme, are presented. Neither patient had a long-standing neurologic disorder, and morphologic evidence of nervous system glucocerebroside storage was
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