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Merck
CN

CDS007521

3-氨基-丙腈

AldrichCPR

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经验公式(希尔记法):
C3H6N2
分子量:
70.09
UNSPSC Code:
12352200
PubChem Substance ID:
EC Number:
205-786-0
MDL number:
NACRES:
NA.21
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InChI key

AGSPXMVUFBBBMO-UHFFFAOYSA-N

InChI

1S/C3H6N2/c4-2-1-3-5/h1-2,4H2

SMILES string

NCCC#N

form

liquid

Other Notes

请注意,Sigma-Aldrich将此产品作为一系列独特化学品的一部分提供给早期发现研究人员。 Sigma-Aldrich不会收集该产品的分析数据。 买方需承担确认产品标识和/或纯度的责任。所有销售均为最终销售。

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signalword

Danger

Hazard Classifications

Acute Tox. 4 Oral - Eye Dam. 1 - Repr. 2 - Skin Corr. 1C

存储类别

8A - Combustible corrosive hazardous materials

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable


历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Brian L Hoh et al.
Journal of the American Heart Association, 7(8) (2018-04-15)
Estrogen deficiency is associated with the development of cerebral aneurysms; however, the mechanism remains unknown. We explored the pathway of cerebral aneurysm development by investigating the potential link between estrogen deficiency and inflammatory factors. First, we established the role of
Taichi Ikedo et al.
World neurosurgery, 129, e361-e366 (2019-06-09)
To investigate the association between vascular morphology and the development of intracranial aneurysms (IAs), the morphological changes of intracranial arteries after IA induction were examined using a rodent model. The vascular morphology of the circle of Willis in rats was
Hirokazu Koseki et al.
Translational stroke research, 11(1), 80-92 (2019-02-10)
Intracranial aneurysm (IA) usually induced at a bifurcation site of intracranial arteries causes a lethal subarachnoid hemorrhage. Currently, IA is considered as a macrophage-mediated inflammatory disease triggered by a high wall shear stress (WSS) on endothelial cells. However, considered the
Haruka Miyata et al.
Journal of neurosurgery, 1-11 (2019-08-17)
Subarachnoid hemorrhage (SAH) has a poor outcome despite modern advancements in medical care. The development of a novel therapeutic strategy to prevent rupture of intracranial aneurysms (IAs) or a novel diagnostic marker to predict rupture-prone lesions is thus mandatory. Therefore
Miyuki Fukuda et al.
Journal of neurosurgery, 1-13 (2019-12-21)
There are no effective therapeutic drugs for cerebral aneurysms, partly because the pathogenesis remains unresolved. Chronic inflammation of the cerebral arterial wall plays an important role in aneurysm formation, but it is not clear what triggers the inflammation. The authors

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