P51
棕榈酸
90%
别名:
C16:0, NSC 5030, 十六酸, 棕榈酸
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关于此项目
线性分子式:
CH3(CH2)14COOH
化学文摘社编号:
分子量:
256.42
EC Number:
200-312-9
UNSPSC Code:
12352100
PubChem Substance ID:
Beilstein/REAXYS Number:
607489
MDL number:
vapor pressure
10 mmHg ( 210 °C)
assay
90%
bp
271.5 °C/100 mmHg (lit.)
mp
61-62.5 °C (lit.)
density
0.852 g/mL at 25 °C (lit.)
SMILES string
CCCCCCCCCCCCCCCC(O)=O
InChI
1S/C16H32O2/c1-2-3-4-5-6-7-8-9-10-11-12-13-14-15-16(17)18/h2-15H2,1H3,(H,17,18)
InChI key
IPCSVZSSVZVIGE-UHFFFAOYSA-N
Gene Information
human ... FABP2(2169), FABP3(2170), FABP4(2167), FABP5(2171)
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存储类别
11 - Combustible Solids
wgk
nwg
flash_point_f
235.4 °F
flash_point_c
113 °C
ppe
dust mask type N95 (US), Eyeshields, Gloves
法规信息
新产品
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B Vessby et al.
Diabetologia, 37(10), 1044-1050 (1994-10-01)
Recent data indicate that peripheral insulin sensitivity may be influenced by dietary fat quality and skeletal muscle phospholipid fatty acid composition. During a health survey of 70-year-old men insulin sensitivity was measured by the euglycaemic hyperinsulinaemic clamp technique and the
Jonas Sieber et al.
The American journal of pathology, 183(3), 735-744 (2013-07-23)
Type 2 diabetes mellitus is characterized by dyslipidemia with elevated free fatty acids (FFAs). Loss of podocytes is a hallmark of diabetic nephropathy, and podocytes are highly susceptible to saturated FFAs but not to protective, monounsaturated FFAs. We report that
Hjalti Kristinsson et al.
Endocrinology, 154(11), 4078-4088 (2013-09-17)
Free fatty acids (FFAs) have pleiotropic effects on the pancreatic β-cell. Although acute exposure to FFAs stimulates glucose-stimulated insulin secretion (GSIS), prolonged exposure impairs GSIS and causes apoptosis. FFAs exert their effects both via intracellular metabolism and interaction with the
Sanja Blaskovic et al.
The FEBS journal, 280(12), 2766-2774 (2013-04-05)
S-palmitoylation is post-translational modification, which consists in the addition of a C16 acyl chain to cytosolic cysteines and which is unique amongst lipid modifications in that it is reversible. It can thus, like phosphorylation or ubiquitination, act as a switch.
Siri Malmgren et al.
The Journal of biological chemistry, 288(17), 11973-11987 (2013-03-12)
Lipotoxicity is a presumed pathogenetic process whereby elevated circulating and stored lipids in type 2 diabetes cause pancreatic β-cell failure. To resolve the underlying molecular mechanisms, we exposed clonal INS-1 832/13 β-cells to palmitate for 48 h. We observed elevated
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