04-921
抗-GluR6/7抗体,克隆NL9,兔单克隆
culture supernatant, clone NL904, Upstate®
别名:
Anti-EAA4, Anti-GLR6, Anti-GLUK6, Anti-GLUR6, Anti-GluK2, Anti-MRT6, Anti-NEDLAS
生物来源
rabbit
质量水平
偶联物
unconjugated
抗体形式
culture supernatant
抗体产品类型
primary antibodies
克隆
NL904, monoclonal
种属反应性
mouse, rat, human
制造商/商品名称
Upstate®
技术
immunoprecipitation (IP): suitable
western blot: suitable
同位素/亚型
IgG
NCBI登记号
UniProt登记号
运输
dry ice
靶向翻译后修饰
unmodified
基因信息
human ... GRIK2(2898)
一般描述
115 kDa
免疫原
KLH偶联的合成肽,对应于大鼠GluR6的氨基酸894-908(C端)。 免疫序列在人中是相同的。
应用
抗GluR6/7抗体,克隆NL9,是一种针对GluR6/7抗体,可用于IP & WB。
研究子类别
神经递质 & 受体
神经递质 & 受体
研究类别
神经科学
神经科学
生化/生理作用
可识别GluR6/7。
外形
无
制备说明
自收到之日起在-20°C可稳定保存2年。避免反复冻融。为了最大程度地回收产品,在融化后和取下盖子之前,将原始样品瓶进行离心。
分析说明
对照
小鼠脑组织、大鼠脑微粒体制剂、人脑裂解液
小鼠脑组织、大鼠脑微粒体制剂、人脑裂解液
已通过免疫印迹分析进行常规评估。
其他说明
替代:05-921
法律信息
UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany
免责声明
除非我们的产品目录或产品附带的其他公司文档另有说明,否则我们的产品仅供研究使用,不得用于任何其他目的,包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。
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储存分类代码
12 - Non Combustible Liquids
WGK
WGK 1
闪点(°F)
Not applicable
闪点(°C)
Not applicable
Gerti Beliu et al.
Communications biology, 2, 261-261 (2019-07-26)
Genetic code expansion (GCE) technology allows the specific incorporation of functionalized noncanonical amino acids (ncAAs) into proteins. Here, we investigated the Diels-Alder reaction between trans-cyclooct-2-ene (TCO)-modified ncAAs, and 22 known and novel 1,2,4,5-tetrazine-dye conjugates spanning the entire visible wavelength range.
Evidence for a role of glutamate as an efferent transmitter in taste buds.
Vandenbeuch A, Tizzano M, Anderson CB, Stone LM, Goldberg D, Kinnamon SC
BMC Neuroscience null
James A Daniel et al.
eLife, 6 (2017-06-10)
SUMO1-conjugation of proteins at neuronal synapses is considered to be a major post-translational regulatory process in nerve cell and synapse function, but the published evidence for SUMO1-conjugation at synapses is contradictory. We employed multiple genetic mouse models for stringently controlled
Qiu-Ju Zhu et al.
FEBS letters, 586(9), 1259-1264 (2012-04-10)
Protein SUMOylation has been implicated in the pathogenesis of ischemic stroke. However, the underlying mechanisms remain unclear. Here, we found that global brain ischemia evokes a sustained elevation of GluK2 SUMOylation in the rat hippocampal CA1 region. Over-expression of wild-type
Neto auxiliary protein interactions regulate kainate and NMDA receptor subunit localization at mossy fiber-CA3 pyramidal cell synapses.
Wyeth, MS; Pelkey, KA; Petralia, RS; Salter, MW; McInnes, RR; McBain, CJ
The Journal of Neuroscience null
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