5000
Oxaloacetic Acid - CAS 328-42-7 - Calbiochem
Substrate for malate dehydrogenase and oxaloacetate decarboxylase.
别名:
Oxaloacetic Acid - CAS 328-42-7 - Calbiochem, OAA
InChI
1S/C4H4O5/c5-2(4(8)9)1-3(6)7/h1H2,(H,6,7)(H,8,9)
InChI key
KHPXUQMNIQBQEV-UHFFFAOYSA-N
assay
>98% (titration)
form
solid
manufacturer/tradename
Calbiochem®
storage condition
OK to freeze
color
white to off-white
solubility
water: soluble
shipped in
ambient
storage temp.
2-8°C
Quality Level
General description
Substrate for malate dehydrogenase and oxaloacetate decarboxylase.
Preparation Note
Following reconstitution, refrigerate (4°C).
Other Notes
Due to the nature of the Hazardous Materials in this shipment, additional shipping charges may be applied to your order. Certain sizes may be exempt from the additional hazardous materials shipping charges. Please contact your local sales office for more information regarding these charges.
Flint, D.H. 1993. Biochemistry 32, 799.
Legal Information
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
Disclaimer
Toxicity: Corrosive (L)
signalword
Warning
hcodes
Hazard Classifications
Eye Irrit. 2
存储类别
13 - Non Combustible Solids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
Ryoki Nakamura et al.
Communications biology, 3(1), 240-240 (2020-05-18)
Members of the leucine-rich repeat-containing 8 (LRRC8) protein family, composed of the five LRRC8A-E isoforms, are pore-forming components of the volume-regulated anion channel (VRAC). LRRC8A and at least one of the other LRRC8 isoforms assemble into heteromers to generate VRAC
Takeshi Yoshida et al.
Frontiers in cell and developmental biology, 7, 316-316 (2019-12-19)
Infantile-onset Pompe disease (IOPD) is a life-threatening multi-organ disease caused by an inborn defect of lysosomal acid α-glucosidase (GAA), which can degrade glycogen into glucose. Lack of GAA causes abnormal accumulation of glycogen in the lysosomes, particularly in the skeletal
Hiroyuki Kabayama et al.
Nature communications, 11(1), 336-336 (2020-01-19)
Targeting cytoplasmic protein-protein interactions with antibodies remains technically challenging, since antibodies expressed in the cytosol frequently form insoluble aggregates. Existing engineering methods are based on the notion that the estimated net charge at pH 7.4 affects stability; as such, they
Contribution of GABAergic interneurons to amyloid-β plaque pathology in an APP knock-in mouse model.
Heather C Rice et al.
Molecular neurodegeneration, 15(1), 3-3 (2020-01-10)
The amyloid-β (Aβ) peptide, the primary constituent of amyloid plaques found in Alzheimer's disease (AD) brains, is derived from sequential proteolytic processing of the Amyloid Precursor Protein (APP). However, the contribution of different cell types to Aβ deposition has not
Nabila Haji et al.
Molecular autism, 11(1), 29-29 (2020-05-08)
Mutations in TSC1 or TSC2 genes cause tuberous sclerosis complex (TSC), a disorder associated with epilepsy, autism, and intellectual disability. TSC1 and TSC2 are repressors of the mechanistic target of rapamycin complex 1 (mTORC1), a key regulator of protein synthesis.
我们的科学家团队拥有各种研究领域经验，包括生命科学、材料科学、化学合成、色谱、分析及许多其他领域.
联系客户支持