569398
Stearic Acid
≥99% (GC), fatty acid, solid
别名:
Stearic Acid, Octadecanoic Acid
产品名称
Stearic Acid,
SMILES string
[O-]C(=O)CCCCCCCCCCCCCCCCC.[H+]
InChI
1S/C18H36O2/c1-2-3-4-5-6-7-8-9-10-11-12-13-14-15-16-17-18(19)20/h2-17H2,1H3,(H,19,20)
InChI key
QIQXTHQIDYTFRH-UHFFFAOYSA-N
description
Merck USA index - 14, 8804
assay
≥99% (GC)
form
solid
manufacturer/tradename
Calbiochem®
storage condition
OK to freeze
impurities
≤0.5% saponifiable substance, as methyl ester
color
white
solubility
ethanol: soluble
shipped in
ambient
storage temp.
10-30°C
Quality Level
Application
Stearic acid has been used as a light chainfatty acid (LCFA) to study its effects on the dysfunction of tumor-derived exosomelipid-induced dendritic cells.
Biochem/physiol Actions
Cell permeable: no
Product does not compete with ATP.
Reversible: no
Disclaimer
Toxicity: Standard Handling (A)
General description
Stearic Acid is a common fatty acid that isfound as a glycerol ester in most plant and animal fats. It exists athigher concentrations in animal fats than plant fats. Stearic acid is found asa triglyceride in cocoa butter and shea butter. It is used to enrich foodinstead of palmitic acid and trans-fat in the food industry. Stearic acid isapplicable in the production of soaps, detergents, shampoos, shaving creams, andcosmetics.
Legal Information
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
存储类别
11 - Combustible Solids
wgk
nwg
flash_point_f
392.0 °F - Cleveland open cup
flash_point_c
200 °C - Cleveland open cup
Diego Cuartas-Marulanda et al.
Polymers, 14(23) (2022-12-12)
Magnesium (Mg) alloys have great potential in biomedical applications due to their incomparable properties regarding other metals, such as stainless steels, Co-Cr alloys, and titanium (Ti) alloys. However, when Mg engages with body fluids, its degradation rate increases, inhibiting the
Xiaozhe Yin et al.
Cell reports, 33(3), 108278-108278 (2020-10-22)
Dendritic cells (DCs) orchestrate the initiation, programming, and regulation of anti-tumor immune responses. Emerging evidence indicates that the tumor microenvironment (TME) induces immune dysfunctional tumor-infiltrating DCs (TIDCs), characterized with both increased intracellular lipid content and mitochondrial respiration. The underlying mechanism
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