AB5725
Anti-Gephyrin Antibody
Chemicon®, from rabbit
别名:
Anti-GEPH, Anti-GPH, Anti-GPHRYN, Anti-HKPX1, Anti-MOCODC
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关于此项目
UNSPSC Code:
12352203
NACRES:
NA.41
eCl@ss:
32160702
Clone:
polyclonal
Species reactivity:
mouse, rat
Application:
WB
Citations:
4
biological source
rabbit
antibody form
affinity purified immunoglobulin
antibody product type
primary antibodies
clone
polyclonal
purified by
affinity chromatography
species reactivity
mouse, rat
manufacturer/tradename
Chemicon®
technique(s)
western blot: suitable
NCBI accession no.
UniProt accession no.
shipped in
wet ice
target post-translational modification
unmodified
Gene Information
human ... GPHN(10243)
Immunogen
Synthetic peptide from rat gephyrin.
Application
Anti-Gephyrin Antibody is an antibody against Gephyrin for use in WB.
Research Category
Neuroscience
Neuroscience
Research Sub Category
Synapse & Synaptic Biology
Synapse & Synaptic Biology
Western blot: 0.5-1.0 μg/mL using ECL on mouse cerebellum tissue lysate. The antibody reacts with a protein of ~93 kDa. An additional band of ~35 kDa may also be seen depending on sample used.
Optimal working dilutions must be determined by the end user.
Optimal working dilutions must be determined by the end user.
Biochem/physiol Actions
Gephyrin
Physical form
Affinity purified immunoglobulin. Liquid.
Preparation Note
Maintain at +2-8°C in undiluted aliquots for up to 6 months after date of receipt.
Other Notes
Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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存储类别
10 - Combustible liquids
wgk
WGK 2
Dibyadeep Datta et al.
Journal of neuroinflammation, 17(1), 8-8 (2020-01-08)
Cognitive impairment in schizophrenia, aging, and Alzheimer's disease is associated with spine and synapse loss from the dorsolateral prefrontal cortex (dlPFC) layer III. Complement cascade signaling is critical in driving spine loss and disease pathogenesis. Complement signaling is initiated by
Yvonne M Ulrich-Lai et al.
Brain structure & function, 221(6), 3141-3156 (2015-08-08)
A history of eating highly palatable foods reduces physiological and emotional responses to stress. For instance, we have previously shown that limited sucrose intake (4 ml of 30 % sucrose twice daily for 14 days) reduces hypothalamic-pituitary-adrenocortical (HPA) axis responses to stress. However
Wen Zhou et al.
International journal of molecular medicine, 38(5), 1481-1489 (2016-12-28)
The medial vestibular nucleus (MVN) and the cerebellar flocculus have been known to be the key areas involved in vestibular compensation (VC) following unilateral labyrinthectomy (UL). In this study, we examined the role of gephyrin and glycine receptor (GlyR) in VC using Sprague-Dawley rats
Jessica S Nikitczuk et al.
Hippocampus, 24(8), 943-962 (2014-04-23)
N-Cadherin and β-catenin form a transsynaptic adhesion complex required for spine and synapse development. In adulthood, N-cadherin mediates persistent synaptic plasticity, but whether the role of N-cadherin at mature synapses is similar to that at developing synapses is unclear. To
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