biological source
rabbit
conjugate
unconjugated
antibody form
affinity purified immunoglobulin
antibody product type
primary antibodies
clone
polyclonal
purified by
affinity chromatography
species reactivity
mouse, human
manufacturer/tradename
Chemicon®
technique(s)
ChIP: suitable, immunohistochemistry: suitable, western blot: suitable
NCBI accession no.
UniProt accession no.
shipped in
dry ice
target post-translational modification
unmodified
Quality Level
Gene Information
human ... RBPJ(3516)
Immunogen
Synthetic peptide, amino acids 194-206 from human RBP-Jk.
Application
Anti-RBP-Jk Antibody is an antibody against RBP-Jk for use in WB, IH.
Biochem/physiol Actions
Recognizes RBP-Jk / RBP-L transcription factor.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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存储类别
12 - Non Combustible Liquids
wgk
WGK 2
flash_point_f
Not applicable
flash_point_c
Not applicable
Histone demethylase KDM5A is an integral part of the core Notch-RBP-J repressor complex.
Liefke, R; Oswald, F; Alvarado, C; Ferres-Marco, D; Mittler, G; Rodriguez, P; Dominguez et al.
Genes & Development null
Eric W Brunskill et al.
Journal of the American Society of Nephrology : JASN, 22(12), 2213-2225 (2011-10-29)
Renin-expressing cells modulate BP, fluid-electrolyte homeostasis, and kidney development, but remarkably little is known regarding the genetic regulatory network that governs the identity of these cells. Here we compared the gene expression profiles of renin cells with most cells in
Olga Gonzalez-Lopez et al.
The Journal of biological chemistry, 294(35), 13073-13092 (2019-07-17)
Reactivation of Kaposi's sarcoma-associated herpesvirus (KSHV) from latency requires the viral transactivator Rta to contact the host protein Jκ recombination signal-binding protein (RBP-Jκ or CSL). RBP-Jκ normally binds DNA sequence-specifically to determine the transcriptional targets of the Notch-signaling pathway, yet
Marie Cibois et al.
RNA (New York, N.Y.), 16(1), 10-15 (2009-11-26)
Targeted inactivations of RNA-binding proteins (RNA-BPs) can lead to huge phenotypical defects. These defects are due to the deregulation of certain mRNAs. However, we generally do not know, among the hundreds of mRNAs that are normally controlled by one RNA-BP
Shinya Ohashi et al.
Gastroenterology, 139(6), 2113-2123 (2010-08-31)
The Notch receptor family regulates cell fate through cell-cell communication. CSL (CBF-1/RBP-jκ, Su(H), Lag-1) drives canonical Notch-mediated gene transcription during cell lineage specification, differentiation, and proliferation in the hematopoietic system, the intestine, the pancreas, and the skin. However, the functional
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