MAB1548
Anti-Myosin Antibody, heavy chain β
culture supernatant, clone 5B9 (2C8), Chemicon®
别名:
Anti-CMD1S, Anti-MPD1, Anti-MYHCB, Anti-SPMD, Anti-SPMM
生物来源
mouse
质量水平
偶联物
unconjugated
抗体形式
culture supernatant
抗体产品类型
primary antibodies
克隆
5B9 (2C8), monoclonal
种属反应性
human
制造商/商品名称
Chemicon®
技术
immunohistochemistry: suitable
western blot: suitable
同位素/亚型
IgG2a
NCBI登记号
UniProt登记号
运输
wet ice
靶向翻译后修饰
unmodified
基因信息
rat ... Myh7(29557)
免疫原
Epitope: heavy chain beta
Human ventricular myosin
应用
Detect Myosin using this Anti-Myosin Antibody, heavy chain β validated for use in WB, IH.
Immunohistochemistry: Neat (undiluted)
Western blot: 1:10
Optimal dilutions must be determined by the end user.
Western blot: 1:10
Optimal dilutions must be determined by the end user.
生化/生理作用
Recognizes the S1/S2 junction of human ventricular myosin heavy chain beta
外形
Supernatant in RPMI 1640, 10% FCS, 0.01% sodium azide.
制备说明
Maintain at -20°C in undiluted aliquots for up to 12 months. Avoid repeated freeze/thaw cycles.
法律信息
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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储存分类代码
12 - Non Combustible Liquids
WGK
WGK 2
闪点(°F)
Not applicable
闪点(°C)
Not applicable
Henry E Young et al.
The anatomical record. Part A, Discoveries in molecular, cellular, and evolutionary biology, 277(1), 178-203 (2004-02-26)
Undifferentiated cells have been identified in the prenatal blastocyst, inner cell mass, and gonadal ridges of rodents and primates, including humans. After isolation these cells express molecular and immunological markers for embryonic cells, capabilities for extended self-renewal, and telomerase activity.
Henry E Young et al.
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Development of a multicellular organism is accomplished through a series of events that are preprogrammed in the genome. These events encompass cellular proliferation, lineage commitment, lineage progression, lineage expression, cellular inhibition, and regulated apoptosis. The sequential progression of cells through
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American journal of physiology. Heart and circulatory physiology, 304(12), H1651-H1661 (2013-04-16)
Ventricular arrhythmias account for high mortality in cardiopulmonary patients in intensive care units. Cardiovascular alterations and molecular-level changes in response to the commonly used oxygen treatment remains unknown. In the present study we investigated cardiac hypertrophy and cardiac complications in
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