产品名称
Anti-β Galactosidase Antibody, clone DC1-4C7, ascites fluid, clone DC1-4C7, Chemicon®
biological source
mouse
antibody form
ascites fluid
antibody product type
primary antibodies
clone
DC1-4C7, monoclonal
species reactivity
E. coli, vertebrates
manufacturer/tradename
Chemicon®
technique(s)
ELISA: suitable
western blot: suitable
isotype
IgG1
NCBI accession no.
UniProt accession no.
shipped in
dry ice
target post-translational modification
unmodified
Gene Information
human ... GLB1(2720)
Application
Anti-β Galactosidase Antibody, clone DC1-4C7 is an antibody against β Galactosidase for use in ELISA & WB.
Research Category
Epitope Tags & General Use
Epitope Tags & General Use
Research Sub Category
Epitope Tags
Epitope Tags
Western blot
ELISA
Optimal working dilutions must be determined by the end user.
ELISA
Optimal working dilutions must be determined by the end user.
Biochem/physiol Actions
Beta galactosidase.
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
Immunogen
Beta galactosidase from E. coli.
Other Notes
Replaces: MAB1802
Physical form
Liquid. Contains no preservative.
Preparation Note
Maintain at -20°C in undiluted aliquots up to 6 months after date of receipt. Avoid repeated freeze/thaw cycles.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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存储类别
10 - Combustible liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
Functional and topological analysis of the Burkholderia cenocepacia priming glucosyltransferase BceB, involved in the biosynthesis of the cepacian exopolysaccharide.
Videira, PA; Garcia, AP; Sa-Correia, I
Journal of Bacteriology null
Eran Perlson et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 29(31), 9903-9917 (2009-08-07)
Retrograde axonal transport of cellular signals driven by dynein is vital for neuronal survival. Mouse models with defects in the retrograde transport machinery, including the Loa mouse (point mutation in dynein) and the Tg(dynamitin) mouse (overexpression of dynamitin), exhibit mild
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