MAB5478
Anti-Retinoid X Receptor α Antibody
ascites fluid, Chemicon®
别名:
RXRalpha
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关于此项目
UNSPSC Code:
12352203
NACRES:
NA.41
eCl@ss:
32160702
Conjugate:
unconjugated
Clone:
monoclonal
Application:
ELISA, EMSA, IP, WB
Citations:
1
biological source
mouse
conjugate
unconjugated
antibody form
ascites fluid
antibody product type
primary antibodies
clone
monoclonal
species reactivity
human, mouse
manufacturer/tradename
Chemicon®
technique(s)
ELISA: suitable, electrophoretic mobility shift assay: suitable, immunoprecipitation (IP): suitable, western blot: suitable
isotype
IgG1
NCBI accession no.
UniProt accession no.
shipped in
wet ice
target post-translational modification
unmodified
Gene Information
human ... RXRA(6256)
Immunogen
Fusion protein corresponding to the DE-region of mouse RXRa.
Application
Use Anti-Retinoid X Receptor α Antibody (Mouse Monoclonal Antibody) validated in ELISA, EMSA, IP, WB to detect Retinoid X Receptor α also known as RXR alpha.
Western blot (endogenous and recombinant protein): 1:500-1:5,000
Immunocytochemistry (transfected cells): 1:500-1:5,000
Immunoprecipitation (endogenous and recombinant protein): 1:500-1:5,000
Gel shift (endogenous and recombinant protein): 1:500-1:5,000
ELISA: 1:500-1:5,000
Optimal working dilutions must be determined by end user.
Immunocytochemistry (transfected cells): 1:500-1:5,000
Immunoprecipitation (endogenous and recombinant protein): 1:500-1:5,000
Gel shift (endogenous and recombinant protein): 1:500-1:5,000
ELISA: 1:500-1:5,000
Optimal working dilutions must be determined by end user.
Biochem/physiol Actions
Reacts with Retinoid X Receptor alpha (RXRalpha). No cross reactivity with RXRbeta or RXRgamma.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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存储类别
10 - Combustible liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
Retinoid-X-receptors (?/?) in melanocytes modulate innate immune responses and differentially regulate cell survival following UV irradiation.
Coleman, DJ; Garcia, G; Hyter, S; Jang, HS; Chagani, S; Liang, X; Larue, L; Ganguli-Indra et al.
PLoS Genetics null
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