等级
JIS special grade
方案
≥98.0%
表单
solid
存货情况
available only in Japan
pH值（酸碱度）
11.5 (20 °C, 20 g/L)
mp
634 °C (lit.)
SMILES字符串
[K]C#N
InChI
1S/CN.K/c1-2;
InChI key
YUZRZFQHUCKACF-UHFFFAOYSA-N
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警示用语：
Danger
危险分类
Acute Tox. 1 Oral - Acute Tox. 2 Dermal - Acute Tox. 2 Inhalation - Aquatic Acute 1 - Aquatic Chronic 1 - Met. Corr. 1 - STOT RE 1
靶器官
Thyroid
补充剂危害
储存分类代码
6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
新产品
此项目有
P Lawson-Smith et al.
Undersea & hyperbaric medicine : journal of the Undersea and Hyperbaric Medical Society, Inc, 38(4), 223-237 (2011-09-01)
Cyanide (CN) intoxication inhibits cellular oxidative metabolism and may result in brain damage. Hydroxycobalamin (OHCob) is one among other antidotes that may be used following intoxication with CN. Hyperbaric oxygen (HBO2) is recommended when supportive measures or antidotes fail. However
Naresh K Sharma et al.
Bioresource technology, 121, 263-273 (2012-08-04)
Present study focused on the degradation of a mixture of phenol, cresol, xylenol, quinoline, and indole along with cyanide, commonly found in coke oven wastewater, using aerobic mixed culture. It was found that xylenol and indole were difficult to degrade
Xiao-Zhang Yu et al.
Journal of hazardous materials, 225-226, 190-194 (2012-05-29)
A study was conducted to investigate activities of nitrate reductase (NR) and glutamine synthetase (GS) in plants during cyanide metabolism. Young rice seedlings (Oryza sativa L. cv. XZX 45) were grown in the nutrient solutions containing KNO(3) or NH(4)Cl and
Vikhyat S Bebarta et al.
Annals of emergency medicine, 64(6), 612-619 (2014-04-22)
Hydroxocobalamin is a Food and Drug Administration-approved antidote for cyanide poisoning. Cobinamide is a potential antidote that contains 2 cyanide-binding sites. To our knowledge, no study has directly compared hydroxocobalamin with cobinamide in a severe, cyanide-toxic large-animal model. Our objective
Sónia C Correia et al.
Neurobiology of disease, 45(1), 206-218 (2011-08-23)
The current study was undertaken to address the role of mitochondrial reactive oxygen species (ROS), and hypoxia inducible factor-1 alpha (HIF-1α) signaling pathway in the protection against high glucose levels in brain endothelial and NT2 neuron-like cells. Rat brain endothelial
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