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Merck
CN

18951

1-异硫氰酸萘酯

derivatization grade (HPLC), LiChropur, ≥98.5% (HPLC and GC)

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线性分子式:
C10H7NCS
化学文摘社编号:
分子量:
185.24
NACRES:
NA.22
PubChem Substance ID:
UNSPSC Code:
41116105
EC Number:
208-990-8
MDL number:
Assay:
≥98.5% (HPLC and GC)
Form:
solid
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InChI key

JBDOSUUXMYMWQH-UHFFFAOYSA-N

InChI

1S/C11H7NS/c13-8-12-11-7-3-5-9-4-1-2-6-10(9)11/h1-7H

SMILES string

S=C=Nc1cccc2ccccc12

grade

derivatization grade (HPLC)

assay

≥98.5% (HPLC and GC)

form

solid

quality

LiChropur

technique(s)

HPLC: suitable

mp

55.5-57 °C (lit.)

Quality Level

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Other Notes

探索最适于 HPLCLC-MS 分析的LiChropur试剂

Legal Information

LiChropur is a trademark of Merck KGaA, Darmstadt, Germany

pictograms

Skull and crossbonesHealth hazard

signalword

Danger

Hazard Classifications

Acute Tox. 3 Oral - Eye Irrit. 2 - Resp. Sens. 1 - Skin Irrit. 2 - STOT SE 3

target_organs

Respiratory system

存储类别

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

危险化学品
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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Igor Sukhotnik et al.
Pediatric surgery international, 28(2), 161-169 (2011-10-13)
Progressive hyperbilirubinemia and end-stage liver failure are among the most serious complications of short bowel syndrome (SBS), representing the principle cause of death in a majority of fatal cases. In the current study, we examined the effects of alpha-naphthylisothiocyanate (ANIT)-induced
Fan Yang et al.
Journal of ethnopharmacology, 144(3), 599-604 (2012-10-13)
To evaluate the hepatoprotective effect of the root and rhizome of Rhubarb (Rhei Radix et Rhizoma) against α-naphthylisothiocyanate (ANIT)-induced liver injury using metabolic profile of bile acids (BAs) along with biochemical parameters and histological alterations. Ultra-performance liquid chromatography coupled with
James P Luyendyk et al.
Toxicological sciences : an official journal of the Society of Toxicology, 119(1), 233-243 (2010-10-27)
Alpha-naphthylisothiocyanate (ANIT)-induced cholestatic liver injury causes tissue factor (TF)-dependent coagulation in mice, and TF deficiency reduces ANIT-induced liver injury. However, the mechanism whereby TF contributes to hepatotoxicity in this model is not known. Utilizing pharmacological and genetic strategies, we evaluated
Bhavana S Solanky et al.
Toxicological sciences : an official journal of the Society of Toxicology, 126(2), 306-316 (2012-01-21)
Certain human diseases affecting the biliary tree can be modeled in rats by ingestion of the hepatobiliary toxin alpha-naphthylisothiocyanate (ANIT). Phosphorus magnetic resonance spectroscopy (MRS) allows the noninvasive monitoring of cell dynamics through detection of phosphodiesters (PDE) and phosphomonoesters (PME).
Juhyun Park et al.
African journal of traditional, complementary, and alternative medicines : AJTCAM, 8(5 Suppl), 220-225 (2011-01-01)
This study evaluated the potential beneficial effect of Moutan Cortex Radicis (MCR) in a murine model of carbon tetrachloride (CCl(4))-, D-galactosamine (GalN)- and α-naphthylisothiocyanate (ANIT)-induced liver injury. Acute hepatotoxicity was induced by intraperitoneal injection of CCl(4) (10 µL/kg), GalN (700

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