产品名称
铜, ACS reagent, granular, 10-40 mesh, ≥99.90%
InChI key
RYGMFSIKBFXOCR-UHFFFAOYSA-N
InChI
1S/Cu
SMILES string
[Cu]
grade
ACS reagent
assay
≥99.90%
form
granular
reaction suitability
core: copper
reagent type: catalyst
resistivity
1.673 μΩ-cm, 20°C
impurities
≤0.001% P
≤0.02% insol. dil. HNO3
particle size
10-40 mesh
bp
2567 °C (lit.)
mp
1083.4 °C (lit.)
density
8.94 g/mL at 25 °C (lit.)
cation traces
Ag: ≤0.002%
As: ≤5 ppm
Fe: ≤0.005%
Mn: ≤0.001%
Pb: ≤0.005%
Sb and Sn: ≤0.01%
Quality Level
Application
Copper (granular) can be used as a catalyst in the hydrogenation of ethylene. It can be impregnated on activated carbon material and used as an adsorbent and also as a catalyst for the removal of SO2 from flue gases.
存储类别
11 - Combustible Solids
wgk
WGK 2
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type P3 (EN 143) respirator cartridges
Carbon materials as catalyst supports for SO2 oxidation: catalytic activity of CuO-AC
Tseng H-H, et al.
Carbon, 41(1), 139-149 (2003)
Effect of occluded hydrogen on the hydrogenation of ethylene over copper
M. C. Schachter, et al.
The Journal of Physical Chemistry, 79(16), 1698?1702-1698?1702 (1975)
Hiroshi Sato et al.
Science (New York, N.Y.), 343(6167), 167-170 (2013-12-18)
Carbon monoxide (CO) produced in many large-scale industrial oxidation processes is difficult to separate from nitrogen (N2), and afterward, CO is further oxidized to carbon dioxide. Here, we report a soft nanoporous crystalline material that selectively adsorbs CO with adaptable
Magnus Andersson et al.
Nature structural & molecular biology, 21(1), 43-48 (2013-12-10)
Heavy metals in cells are typically regulated by PIB-type ATPases. The first structure of the class, a Cu(+)-ATPase from Legionella pneumophila (LpCopA), outlined a copper transport pathway across the membrane, which was inferred to be occluded. Here we show by
Yan Meng et al.
Biochimica et biophysica acta, 1690(3), 208-219 (2004-10-30)
Hepatic abnormalities in Long-Evans Cinnamon (LEC) rats, an animal model of Wilson disease (WD), were restored by the expression of the human ATP7B cDNA under the control of CAG promoter. Expression of ATP7B transcript and protein in the liver of
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