等级
purum
方案
≥98.0% (HPLC)
mp
118-122 °C (lit.)
118-122 °C
SMILES字符串
O=C1C(c2ccccc2)=C1c3ccccc3
InChI
1S/C15H10O/c16-15-13(11-7-3-1-4-8-11)14(15)12-9-5-2-6-10-12/h1-10H
InChI key
HCIBTBXNLVOFER-UHFFFAOYSA-N
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Warning
危险声明
危险分类
Skin Sens. 1
储存分类代码
11 - Combustible Solids
WGK
WGK 3
个人防护装备
dust mask type N95 (US), Eyeshields, Faceshields, Gloves
法规信息
新产品
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Leena M Koulu et al.
The Journal of investigative dermatology, 130(11), 2578-2582 (2010-07-09)
Polymorphic light eruption (PLE) is a common skin disorder provoked by exposure to UVR. Its clinical symptoms resemble those of a contact allergic reaction. PLE is generally considered a T-cell-mediated autoimmune reaction toward a yet unidentified antigen formed in UVR-exposed
N Bangsgaard et al.
Clinical and experimental immunology, 165(3), 310-317 (2011-06-15)
An inverse relation between contact allergy and autoimmune diseases is suggested from epidemiological studies. The aim of this study was to investigate susceptibility and reactivity in patients with psoriasis, patients with diabetes and healthy controls in an experimental sensitization study.
Morihiko Hirota et al.
The Journal of toxicological sciences, 35(6), 871-879 (2010-12-09)
Changes of cell-surface thiols induced by chemical treatment may affect the conformations of membrane proteins and intracellular signaling mechanisms. In our previous study, we found that a non-toxic dose of diphenylcyclopropene (DPCP), which is a potent skin sensitizer, induced an
Shigeki Inui et al.
The Journal of dermatology, 37(10), 888-893 (2010-09-24)
Pigmentary complication by contact immunotherapy (CI) for alopecia areata (AA) has been reported but its pathophysiology remains unknown. To characterize pigmentary complication by CI and its pathophysiology, we examined the incidence of hyperpigmentation in 186 consecutive patients treated with CI
Nahla Hunter et al.
The Journal of dermatological treatment, 22(1), 2-10 (2010-06-08)
To evaluate the efficacy of two topically applied immunomodulative agents through the detection of lymphocyte subsets using monoclonal antibodies against CD4, CD8 and MHC II. Fifty patients from the Departments of Medical Biochemistry, Dermatology and Pathology at Cairo University with
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