InChI key
HOVAGTYPODGVJG-PZRMXXKTSA-N
InChI
1S/C7H14O6/c1-12-7-6(11)5(10)4(9)3(2-8)13-7/h3-11H,2H2,1H3/t3-,4+,5+,6-,7+/m1/s1
SMILES string
CO[C@H]1O[C@H](CO)[C@H](O)[C@H](O)[C@H]1O
grade
purum
assay
≥98.0% (sum of enantiomers, HPLC)
optical activity
[α]20/D +169±3°, c = 1.5% in methanol
impurities
~8.5% water
mp
116-117 °C (lit.)
存储类别
13 - Non Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
法规信息
新产品
此项目有
T J Kelley et al.
Bioscience reports, 19(5), 433-447 (2000-04-14)
The highly purified DNA Pol-alpha from rat prostate tumor (PA-3) and human neuroblastoma (IMR-32) cells appeared to be inhibited by Ricin (RCA-II), and Con-A. Loss of activity (40 to 60%) of a specific form of DNA polymerase from IMR-32 was
Alexander I Zinin et al.
Carbohydrate research, 337(7), 635-642 (2002-03-23)
1-O-Acetyl-beta-D-galactopyranose (AcGal), a new substrate for beta-galactosidase, was synthesized in a stereoselective manner by the trichloroacetimidate procedure. Kinetic parameters (K(M) and k(cat)) for the hydrolysis of 1-O-acetyl-beta-D-galactopyranose catalyzed by the beta-D-galactosidase from Penicillium sp. were compared with similar characteristics for
Yan Yang et al.
Carbohydrate research, 342(8), 1063-1070 (2007-03-16)
PIP60-1, a novel heteropolysaccharide isolated from fruiting bodies of the medicinal fungus, Phellinus igniarius, has a molecular weight of 1.71 x 10(4)Da and is composed of L-fucose, D-glucose, D-mannose, D-galactose and 3-O-Me-D-galactose in a ratio of 1:1:1:2:1. A structural investigation
N Declerck et al.
Protein engineering, 7(8), 997-1004 (1994-08-01)
The L-arabinose binding protein (ABP) of Escherichia coli naturally binds L-arabinose and D-galactose with very high affinity and, with reduced affinity, a variety of other sugars that differ only at the C5 position of the pyranose ring. However, there are
Satoru Takahashi et al.
Epilepsy research, 80(1), 18-22 (2008-05-06)
Glucose transporter 1 (GLUT1) deficiency syndrome is caused by a deficit in glucose transport to the brain during the pre- and postnatal periods. Here, we report two cases of GLUT1 deficiency syndrome diagnosed on the basis of clinical features, reduced
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