81720
DL-脯氨酸
purum, ≥98.0% (NT)
别名:
(±)-吡咯烷-2-羧酸
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关于此项目
经验公式（希尔记法）:
C5H9NO2
化学文摘社编号:
分子量:
115.13
EC Number:
210-189-3
UNSPSC Code:
12352200
PubChem Substance ID:
Beilstein/REAXYS Number:
80812
MDL number:
grade
purum
assay
≥98.0% (NT)
mp
208 °C (dec.) (lit.), ~210 °C (dec.)
SMILES string
OC(=O)C1CCCN1
InChI
1S/C5H9NO2/c7-5(8)4-2-1-3-6-4/h4,6H,1-3H2,(H,7,8)
InChI key
ONIBWKKTOPOVIA-UHFFFAOYSA-N
存储类别
13 - Non Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
法规信息
新产品
此项目有
Jian Liu et al.
American journal of physiology. Heart and circulatory physiology, 307(4), H598-H608 (2014-06-16)
High-fat, low-carbohydrate diets (HFLCD) are often eaten by humans for a variety of reasons, but the effects of such diets on the heart are incompletely understood. We evaluated the impact of HFLCD on myocardial ischemia/reperfusion (I/R) using an in vivo
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PloS one, 9(6), e98336-e98336 (2014-06-05)
Enzyme replacement therapy (ERT) with recombinant human acid-α-glucosidase (rhGAA) is the only FDA approved therapy for Pompe disease. Without ERT, severely affected individuals (early onset) succumb to the disease within 2 years of life. A spectrum of disease severity and
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Cardiovascular diabetology, 13, 89-89 (2014-06-03)
Despite advances in the treatment of heart failure, mortality remains high, particularly in individuals with diabetes. Activated transforming growth factor beta (TGF-β) contributes to the pathogenesis of the fibrotic interstitium observed in diabetic cardiomyopathy. We hypothesized that high glucose enhances
Sarah C Huen et al.
Pediatric nephrology (Berlin, Germany), 30(2), 199-209 (2014-01-21)
Acute ischemic kidney injury is a common complication in hospitalized patients. No treatment is yet available for augmenting kidney repair or preventing progressive kidney fibrosis. Animal models of acute kidney injury demonstrate that activation of the innate immune system plays
Leontine E H Bakker et al.
Diabetologia, 58(1), 165-177 (2014-10-16)
South Asians have a higher risk of developing type 2 diabetes than Europeans. The underlying cause of this excess risk is still poorly understood but might be related to differences in the regulation of energy/nutrient-sensing pathways in metabolic tissues and
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