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Merck
CN

95000

1,1-二氯乙烯

puriss., ≥99.5% (GC)

别名:

偏二氯乙烯

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关于此项目

线性分子式:
CH2=CCl2
化学文摘社编号:
分子量:
96.94
EC Number:
200-864-0
UNSPSC Code:
12162002
MDL number:
Beilstein/REAXYS Number:
1733365
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vapor density

3.46 (vs air)

vapor pressure

9.68 psi ( 20 °C)

grade

puriss.

assay

≥99.5% (GC)

autoignition temp.

968 °F

contains

~0.02% 4-methoxyphenol as stabilizer

expl. lim.

15.5 %

refractive index

n20/D 1.426

bp

30-32 °C

mp

−122 °C (lit.)

density

1.213 g/mL at 20 °C (lit.)

storage temp.

2-8°C

SMILES string

ClC(Cl)=C

InChI

1S/C2H2Cl2/c1-2(3)4/h1H2

InChI key

LGXVIGDEPROXKC-UHFFFAOYSA-N



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signalword

Danger

Hazard Classifications

Acute Tox. 3 Oral - Acute Tox. 4 Inhalation - Aquatic Chronic 2 - Carc. 2 - Eye Irrit. 2 - Flam. Liq. 1 - STOT RE 1 Inhalation - STOT RE 2 Oral

target_organs

Liver, Nose

存储类别

3 - Flammable liquids

wgk

WGK 3

flash_point_f

-9.4 °F - closed cup

flash_point_c

-23.0 °C - closed cup

ppe

Eyeshields, Faceshields, Gloves

法规信息

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Young Kim et al.
Biotechnology and bioengineering, 80(5), 498-508 (2002-10-02)
Batch kinetic and inhibition studies were performed for the aerobic cometabolism of 1,1,1-trichloroethane (1,1,1-TCA), 1,1-dichloroethylene (1,1-DCE), and 1,1-dichloroethane (1,1-DCA) by a butane-grown mixed culture. These chlorinated aliphatic hydrocarbons (CAHs) are often found together as cocontaminants in groundwater. The maximum degradation
Masarin Ban et al.
Toxicology, 226(2-3), 161-171 (2006-08-02)
Occupational allergy and asthma is a challenging issue in the developing countries. Chemicals inhaled in the workplaces may act not only as allergens but also as immune response modifiers, contributing to asthma exacerbation. In this study, we tested the adjuvant
Erik J Martin et al.
The Journal of pharmacology and experimental therapeutics, 313(1), 95-103 (2005-01-01)
1,1-Dichloroethylene (DCE) causes pulmonary injury that is characterized by necrosis of bronchiolar Clara cells. Mitochondria have been identified as an early target in the toxic response. Because mitochondria have been implicated in both necrotic and apoptotic cell death, we have