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Merck
CN

78319

反-戊烯二酸 钠盐 水合物

≥95.0% (HPLC)

别名:

(2E)-Pentenedioic acid sodium salt hydrate, Sodium trans-glutaconate hydrate

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关于此项目

经验公式(希尔记法):
C5H6O4 · xNa+ · yH2O
分子量:
130.10 (anhydrous free acid basis)
UNSPSC Code:
12352202
PubChem Substance ID:
NACRES:
NA.25
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assay

≥95.0% (HPLC)

form

powder

SMILES string

OC(C/C=C/C(O)=O)=O.C

InChI

1S/C5H6O4.CH4/c6-4(7)2-1-3-5(8)9;/h1-2H,3H2,(H,6,7)(H,8,9);1H4/b2-1+;

InChI key

NRJOWSDIBMLXCK-TYYBGVCCSA-N

Application

Internal reference for urine samples

存储类别

11 - Combustible Solids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

新产品

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分析证书(COA)

Lot/Batch Number

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Metabolomics of urinary organic acids in respiratory chain deficiencies in children.
Reinecke, C.J., et al.
Metabolomics, 8, 264-283 (2012)
Júlia Dénes et al.
Analytical chemistry, 84(22), 10113-10120 (2012-10-26)
A novel, single stage high resolution mass spectrometry-based method is presented for the population level screening of inborn errors of metabolism. The approach proposed here extends traditional electrospray tandem mass spectrometry screening by introducing nanospray ionization and high resolution mass
Atsuki Ikeda et al.
Biomedical chromatography : BMC, 26(5), 548-558 (2011-07-21)
Conventional tumor markers are unsuitable for detecting carcinoma at an early stage and lack clinical efficacy and utility. In this study, we attempted to investigate the differences in serum metabolite profiles of gastrointestinal cancers and healthy volunteers using a metabolomic
G F Hoffmann et al.
Journal of inherited metabolic disease, 22(4), 381-391 (1999-07-17)
The biochemical hallmark of glutaric aciduria type I (GA I) due to glutaryl-CoA dehydrogenase deficiency is the accumulation of glutaric acid, and to a lesser degree of 3-hydroxyglutaric and glutaconic acids. Abnormal metabolites vary from gross organic aciduria to only
Jonathan C Schisler et al.
Journal of the American Heart Association, 4(2), doi:10-doi:10 (2015-02-26)
Long chain acyl-CoA synthetases (ACSL) catalyze long-chain fatty acids (FA) conversion to acyl-CoAs. Temporal ACSL1 inactivation in mouse hearts (Acsl1(H-/-)) impaired FA oxidation and dramatically increased glucose uptake, glucose oxidation, and mTOR activation, resulting in cardiac hypertrophy. We used unbiased

全球贸易项目编号

货号GTIN
78319-100MG04061832860657
78319-10MG04061832860664
78319-500MG04061832860671

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