A0207
(+)-Iron(II) L-ascorbate
≥90% (titration)
别名:
L-(+)-Ascorbic acid iron(II) salt, Ferrous ascorbate, Vitamin C iron(II) salt
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关于此项目
经验公式（希尔记法）:
C12H14FeO12
化学文摘社编号:
分子量:
406.08
NACRES:
NA.79
PubChem Substance ID:
UNSPSC Code:
12352200
EC Number:
246-469-7
MDL number:
assay
≥90% (titration)
form
crystalline
color
dark brown
SMILES string
[H][C@@]1(OC(=O)C(O)=C1O[Fe]OC2=C(O)C(=O)O[C@]2([H])[C@@H](O)CO)[C@@H](O)CO
InChI
1S/2C6H8O6.Fe/c2*7-1-2(8)5-3(9)4(10)6(11)12-5;/h2*2,5,7-10H,1H2;/q;;+2/p-2/t2*2-,5+;/m00./s1
InChI key
RFBYLSCVRUTUSB-ZZMNMWMASA-L
Biochem/physiol Actions
(+)-Iron(II) L-ascorbate or ferrous ascorbate has high potential to absorb iron in vivo compared to ferrous sulfate. In children, it is used as an effective oral iron supplement to treat iron deficiency anemia.
Iron-ascorbate (ferrous-ascorbate) is used as a reagent in cell culture and other systems to evaluate various aspects of oxidative stress and anti-oxidation mechanisms.
Disclaimer
May darken in storage.
存储类别
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
Rame Taha et al.
PloS one, 5(7), e11817-e11817 (2010-08-03)
Although mitochondrial dysfunction and oxidative stress are central mechanisms in various pathological conditions, they have not been extensively studied in the gastrointestinal tract, which is known to be constantly exposed to luminal oxidants from ingested foods. Key among these is
Dirk Hofreuter et al.
PloS one, 7(11), e50699-e50699 (2012-12-12)
Campylobacter jejuni is a major cause of food-borne disease in industrialized countries. Carbohydrate utilization by C. jejuni is severely restricted, and knowledge about which substrates fuel C. jejuni infection and growth is limited. Some amino acids have been shown to
Ingrid Wiswedel et al.
Free radical research, 44(2), 135-145 (2010-01-22)
The aim of this study was to investigate the effect of oxidative stress on mitochondrial phospholipids. In this context, this study investigated (i) the content of phosphatidylethanolamine (PE), phosphatidylcholine (PC) and cardiolipin (CL), (ii) the correlation of CL degradation with