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Merck
CN

A7352

Sigma-Aldrich

Anti-ADAM-33, Propeptide Region antibody produced in rabbit

~1 mg/mL, affinity isolated antibody, buffered aqueous glycerol solution

别名:

Anti-A Disintegrin And Metalloproteinase-33

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MDL编号:
UNSPSC代码:
12352203
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生物来源

rabbit

偶联物

unconjugated

抗体形式

affinity isolated antibody

抗体产品类型

primary antibodies

克隆

polyclonal

表单

buffered aqueous glycerol solution

种属反应性

human

浓度

~1 mg/mL

技术

western blot: 1:1,000

UniProt登记号

运输

wet ice

储存温度

−20°C

靶向翻译后修饰

unmodified

基因信息

human ... ADAM33(80332)

一般描述

A Disintegrin And Metalloproteinase-33 (ADAM-3) is a widely expressed protein that egulates cell-attachment by binding to integrins via cysteine-rich domains. ADAM-33 has also been associated with asthma susceptibility. Rabbit Anti-ADAM-33, Propeptide Region antibody binds to the amino end of human ADAM-33.
By immunoblotting against the reduced protein, the antibody identifies bands at 99 kDa (minor band), 58 kDa (major band), and cleaved products in cell lysates.

免疫原

synthetic peptide corresponding to the amino end of human ADAM-33.

应用

Rabbit Anti-ADAM-33, Propeptide Region antibody can be used for western blot applications at 1:1,000.

外形

Solution in phosphate buffered saline containing 50% glycerol and 0.05% sodium azide.

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储存分类代码

10 - Combustible liquids

WGK

WGK 1

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)

法规信息

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分析证书(COA)

Lot/Batch Number

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Tsuyoshi Yoshinaka et al.
Gene, 282(1-2), 227-236 (2002-01-30)
The ADAM family of membrane-anchored proteins has a unique domain structure, with each containing a disintegrin and metalloprotease (ADAM) domain. We have isolated mouse and human cDNAs encoding a novel member of the ADAM family. The mouse and human predicted
Paul Van Eerdewegh et al.
Nature, 418(6896), 426-430 (2002-07-12)
Asthma is a common respiratory disorder characterized by recurrent episodes of coughing, wheezing and breathlessness. Although environmental factors such as allergen exposure are risk factors in the development of asthma, both twin and family studies point to a strong genetic

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