C6118
Monoclonal Anti- Cdk5 小鼠抗
~2 mg/mL, clone CDK-3G, purified immunoglobulin, buffered aqueous solution
别名:
Anti- Cyclin Dependent Kinase 5, Anti- PSSALRE
biological source
mouse
conjugate
unconjugated
antibody form
purified immunoglobulin
antibody product type
primary antibodies
clone
CDK-3G, monoclonal
form
buffered aqueous solution
mol wt
~34 kDa
species reactivity
mouse, human
concentration
~2 mg/mL
technique(s)
indirect ELISA: suitable, western blot: 2-4 μg/mL using total cell extracts of mouse brain cytosolic fraction S1.
isotype
IgM
UniProt accession no.
shipped in
dry ice
storage temp.
−20°C
Gene Information
human ... CDK5(1020)
mouse ... Cdk5(12568)
Immunogen
synthetic peptide corresponding to amino acids 269-285 of human Cdk5, conjugated to KLH.
Application
Applications in which this antibody has been used successfully, and the associated peer-reviewed papers, are given below.
Immunocytochemistry (1 paper)
Immunocytochemistry (1 paper)
Physical form
0.01M 磷酸缓冲盐溶液，pH 7.4，含 15mM 叠氮化钠。
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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存储类别
12 - Non Combustible Liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)
法规信息
新产品
此项目有
Veronika Reiter et al.
Nucleic acids research, 40(13), 6235-6240 (2012-03-17)
The unusual cyclin-dependent protein kinase 5 (CDK5) was discovered based on its sequence homology to cell cycle regulating CDKs. CDK5 was found to be active in brain tissues, where it is not involved in cell cycle regulation but in the
Viviana Triaca et al.
Aging cell, 15(4), 661-672 (2016-04-15)
NGF has been implicated in forebrain neuroprotection from amyloidogenesis and Alzheimer's disease (AD). However, the underlying molecular mechanisms are still poorly understood. Here, we investigated the role of NGF signalling in the metabolism of amyloid precursor protein (APP) in forebrain
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