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Merck
CN

GW21036

Anti-RANTES antibody produced in chicken

affinity isolated antibody, buffered aqueous solution

别名:

Anti-SCYA5, Anti-Small inducible cytokine A5

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关于此项目

UNSPSC Code:
12352203
MDL number:
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biological source

chicken

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

form

buffered aqueous solution

species reactivity

rat, mouse, human

manufacturer/tradename

Genway 15-288-21036

technique(s)

indirect ELISA: suitable, western blot: suitable

NCBI accession no.

UniProt accession no.

shipped in

wet ice

storage temp.

−20°C

Gene Information

human ... CCL5(6352)

Immunogen

Immunogen Sequence: GI # 4506847, sequence 24-90
Recombinant small inducible cytokine A5 (RANTES); T-cell specific protein p288

Physical form

Solution in phosphate buffered saline containing 0.02% sodium azide.


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存储类别

10 - Combustible liquids

wgk

WGK 1

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)

法规信息

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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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相关内容


Yongfeng Sun et al.
Transplantation, 97(12), 1233-1239 (2014-05-16)
Regulated on activation normal T-cell expressed and secreted (RANTES)-chemokine ligand 5 plays a key role in mediating heart transplant rejection. Suppression of RANTES-mediated signals can reduce leukocyte recruitment and mitigate transplant rejection severity. The present study describes the construction of
Hye Young Kim et al.
Cytokine, 68(1), 40-49 (2014-03-25)
An anti-inflammatory cytokine, interleukin-10 (IL-10) exerts inhibitory effects on vascular inflammation. Chemokines promote vascular inflammation and play a pathogenic role in the development and maintenance of hypertension. However, chemokine CCL5 has down-regulatory effects on angiotensin II (Ang II)-induced hypertensive mediators.
Egle Cekanaviciute et al.
Journal of immunology (Baltimore, Md. : 1950), 193(1), 139-149 (2014-05-27)
The balance between controlling infection and limiting inflammation is particularly precarious in the brain because of its unique vulnerability to the toxic effects of inflammation. Astrocytes have been implicated as key regulators of neuroinflammation in CNS infections, including infection with