size
15 cm × 15 cm
pore size
0.45 μm
SMILES string
OC[C@@H]1O[C@@H](O[C@H]2[C@@H](O)[C@H](O)[C@@H](O)O[C@H]2CO)[C@@H](O)[C@H](O)[C@H]1O.[O-][N+](=O)OC[C@@H]3O[C@H](O[N+]([O-])=O)[C@@H](O[N+]([O-])=O)[C@H](O[N+]([O-])=O)[C@@H]3O[C@H]4O[C@@H](CO[N+]([O-])=O)[C@@H](O[N+]([O-])=O)[C@@H](O[N+]([O-])=O)[C@@H]4O[N+]([O-])=O
Application
For use in dot blots and Southern blots
Agnieszka Mykowska et al.
Nucleic acids research, 39(20), 8938-8951 (2011-07-29)
Mutant transcripts containing expanded CUG repeats in the untranslated region are a pathogenic factor in myotonic dystrophy type 1 (DM1). The mutant RNA sequesters the muscleblind-like 1 (MBNL1) splicing factor and causes misregulation of the alternative splicing of multiple genes
Gerhard Leinenga et al.
Science translational medicine, 7(278), 278ra33-278ra33 (2015-03-13)
Amyloid-β (Aβ) peptide has been implicated in the pathogenesis of Alzheimer's disease (AD). We present a nonpharmacological approach for removing Aβ and restoring memory function in a mouse model of AD in which Aβ is deposited in the brain. We
Sunniva Stordal Bjørklund et al.
BMC cancer, 15, 524-524 (2015-07-18)
Alternate transcripts from a single gene locus greatly enhance the combinatorial flexibility of the human transcriptome. Different patterns of exon usage have been observed when comparing normal tissue to cancers, suggesting that variant transcripts may play a role in the
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