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Merck
CN

PBS1

Sigma-Aldrich

磷酸盐缓冲盐溶液体系

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UNSPSC代码:
12161700
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表单

liquid

溶解性

water: soluble

包装

用于制备定制 PBS 缓冲液的储存液套装

仅试剂盒组分

产品编号
说明

  • 1M KH2PO4 1 L

  • 1M K2HPO4 1 L

  • 5M NaCl 1 L

储存分类代码

10 - Combustible liquids

WGK

WGK 1

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)

法规信息

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分析证书(COA)

Lot/Batch Number

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Kayluz F Boligan et al.
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To explore the repertoire of glycan-specific immunoglobulin G (IgG) antibodies in treatment-naive patients with relapsing-remitting multiple sclerosis (RRMS). A systems-level approach combined with glycan array technologies was used to determine specificities and binding reactivities of glycan-specific IgGs in treatment-naive patients
Laura Millares et al.
International journal of chronic obstructive pulmonary disease, 12, 2807-2811 (2017-10-17)
The bronchial mucosa is protected by a specialized immune system focused on the prevention of colonization and infection by potentially pathogenic microorganisms (PPMs). Immunoglobulin A (IgA) is the principal antibody involved in this mechanism. A defective immune barrier may facilitate
Silvana Ramos Atayde et al.
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Because collagen type V (Col V) can be exposed in tissue injury, we hypothesized that oral administration of this collagen species modulates the inflammation and remodeling of experimental synovitis, avoiding joint destruction, and that the modulation may differ according to
Karen Kerkhof et al.
Malaria journal, 15(1), 529-529 (2016-11-05)
Serological markers for exposure to different Plasmodium species have recently been used in multiplex immunoassays based on the Luminex technology. However, interpretation of the assay results requires consideration of the half-life of specific antibodies against these markers. Therefore, the aim
Stine Spray et al.
Journal of molecular neuroscience : MN, 61(3), 396-411 (2016-12-10)
This study aimed at obtaining an in-depth mapping of expressional changes of the cerebral microvasculature after transient global cerebral ischemia (GCI) and the impact on these GCI-induced expressional changes of post-GCI treatment with a mitogen-activated protein kinase kinase (MEK1/2) inhibitor.

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