SAB1300089
抗GPR81-S296 兔抗
affinity isolated antibody, buffered aqueous solution
别名:
抗FKSG80, 抗GPR104, 抗GPR81, 抗G蛋白偶联受体104, 抗G蛋白偶联受体81
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关于此项目
NACRES:
NA.41
UNSPSC Code:
12352203
Conjugate:
unconjugated
Clone:
polyclonal
Application:
ELISA (i), IHC, WB
Citations:
3
biological source
rabbit
conjugate
unconjugated
antibody form
affinity isolated antibody
antibody product type
primary antibodies
clone
polyclonal
form
buffered aqueous solution
species reactivity
rat, human, mouse
technique(s)
immunohistochemistry: 1:50-1:100, indirect ELISA: 1:1000, western blot: 1:100-1:500
NCBI accession no.
shipped in
dry ice
storage temp.
−20°C
target post-translational modification
unmodified
Gene Information
human ... GPR81(27198)
General description
G蛋白偶联受体（GPCR或GPR）包含7个跨膜结构域，通过异三聚体G蛋白转导细胞外信号。RNA印迹分析揭示了垂体中GPR81的表达情况。
Immunogen
GPR81（NP_115943，286-332）
该抗体是由KLH偶联合成肽免疫的兔产生的，该肽选自人GPR81-S296区域。
该抗体是由KLH偶联合成肽免疫的兔产生的，该肽选自人GPR81-S296区域。
Application
成功使用该抗体的应用以及相关的同行评审论文如下所示。
蛋白质免疫印迹分析（1篇论文）
蛋白质免疫印迹分析（1篇论文）
Physical form
纯化的单克隆抗体是含有0.09％（W/V）叠氮化钠的PBS溶液
Disclaimer
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存储类别
12 - Non Combustible Liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
低风险生物材料
常规特殊物品
此项目有
Xiaojing Li et al.
Molecular nutrition & food research, 67(24), e2300444-e2300444 (2023-10-29)
Epidemiological studies indicate an inverse correlation between yogurt consumption and colorectal cancer (CRC), but whether there is a cause-and-effect relationship has not yet been validated. This study aims to investigate the effects and possible mechanisms of yogurt on colitis-associated colorectal
Haíssa de Castro Abrantes et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 39(23), 4422-4433 (2019-03-31)
The discovery of a G-protein-coupled receptor for lactate named hydroxycarboxylic acid receptor 1 (HCAR1) in neurons has pointed to additional nonmetabolic effects of lactate for regulating neuronal network activity. In this study, we characterized the intracellular pathways engaged by HCAR1
Liangchun Yang et al.
Nature communications, 5, 4436-4436 (2014-07-16)
Increasing evidence suggests the important role of metabolic reprogramming in the regulation of the innate inflammatory response, but the underlying mechanism remains unclear. Here we provide evidence to support a novel role for the pyruvate kinase M2 (PKM2)-mediated Warburg effect
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