跳转至内容
Merck
CN

SAB4503206

Anti-T3JAM antibody produced in rabbit

affinity isolated antibody

别名:

T3JAM, TRAF3 interacting protein 3, TRAF3-interacting JNK-activating modulator, TRAF3-interacting Jun N-terminal kinase (JNK)-activating modulator, TRAF3-interacting protein 3

登录 查看组织和合同定价。

选择尺寸

变更视图

关于此项目

NACRES:
NA.41
UNSPSC Code:
12352203
技术服务
需要帮助?我们经验丰富的科学家团队随时乐意为您服务。
让我们为您提供帮助


biological source

rabbit

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

form

buffered aqueous solution

mol wt

antigen 63 kDa

species reactivity

human, mouse, rat

concentration

~1 mg/mL

technique(s)

ELISA: 1:20000, immunofluorescence: 1:100-1:500, western blot: 1:500-1:1000

NCBI accession no.

UniProt accession no.

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... TRAF3IP3(80342)

General description

Anti-T3JAM Antibody detects endogenous levels of total T3JAM protein.

Immunogen

The antiserum was produced against synthesized peptide derived from human T3JAM.

Immunogen Range: 251-300

Features and Benefits

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

Physical form

Rabbit IgG in phosphate buffered saline (without Mg2+ and Ca2+), pH 7.4, 150mM NaCl, 0.02% sodium azide and 50% glycerol.


Still not finding the right product?

试用我们的 产品选型工具 工具缩小选择范围


存储类别

10 - Combustible liquids

wgk

nwg

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

新产品

此项目有



历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

没有发现合适的版本?

如果您需要特殊版本,可通过批号或批次号查找具体证书。

已有该产品?

在文件库中查找您最近购买产品的文档。

访问文档库



Chin-Yi Cheng et al.
Chinese medicine, 16(1), 82-82 (2021-08-23)
Post-ischemic inflammation is a crucial component in stroke pathology in the early phase of cerebral ischemia-reperfusion (I/R) injury. Inflammation caused by microglia, astrocytes, and necrotic cells, produces pro-inflammatory mediators and exacerbates cerebral I/R injury. This study evaluated the effects of