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NACRES:
NA.41
UNSPSC Code:
12352203
Species reactivity:
vertebrates
Clone:
RM161, monoclonal, recombinant monoclonal
Application:
ChIP, ELISA, ICC, IHC, WB, multiplexing
Citations:
2
species reactivity
vertebrates
recombinant
expressed in HEK 293 cells
antibody form
purified immunoglobulin
antibody product type
primary antibodies
clone
RM161, monoclonal, recombinant monoclonal
form
buffered aqueous glycerol solution
technique(s)
ChIP: 2-10 μg/mL, ELISA: 0.5-2 μg/mL, immunocytochemistry: 0.5-2 μg/mL, immunohistochemistry: 0.1—1 μg/mL, multiplexing: 0.05-0.5 μg/mL, western blot: 0.25— 1 μg/mL
isotype
IgG
NCBI accession no.
UniProt accession no.
shipped in
wet ice
storage temp.
−20°C
target post-translational modification
acetylation (Lys9)
Gene Information
human ... H3C1(8350)
Immunogen
An acetyl-peptide corresponding to the Acetyl-Histone H3 (Lys9)
Biochem/physiol Actions
This antibody reacts to Histone H3 acetylated at Lysine 9 (K9ac). No cross reactivity with other acetylated Lysines in histone H3.
Physical form
Solution in phosphate buffered saline containing 50% Glycerol/PBS with 1% BSA and 0.09% sodium azide.
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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存储类别
10 - Combustible liquids
wgk
WGK 2
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
低风险生物材料
常规特殊物品
此项目有
Lin Xiao et al.
Clinical epigenetics, 13(1), 53-53 (2021-03-12)
Intervertebral disc degeneration (IDD) is a major cause of lower back pain. This study aimed at exploring the effects of histone deacetylase 4 (HDAC4) and its upstream and downstream signaling molecules on IDD development. A murine IDD model was established
Hang Xue et al.
American journal of physiology. Cell physiology, 319(2), C381-C391 (2020-06-04)
Several microRNAs (miRNAs or miRs) regulate cerebral ischemic injury outcomes; however, little is known about the role of miR-539-5p during cerebral ischemic injury or the postischemic state. Cerebral ischemic injury was modeled in vitro by exposing human cortical neurons to
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