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Merck
CN

10342

Amberlite IRN77 氢型

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UNSPSC Code:
23151817
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form

beads

crosslinking

8 % cross-linked

packaging

bucket of 500 g

parameter

121 °C max. temp.

moisture

49-55%

technique(s)

LPLC: suitable

matrix

styrene-divinylbenzene (gel)

matrix active group

SCX

particle size

600-700 μm

operating pH

0-14

capacity

1.9 meq/mL by wetted bed volume

separation technique

cation exchange

General description

作为核级树脂,至少 99% 的有效交换部都以氢形式存在。
强酸性阳离子交换树脂,用于水处理,放射性废弃物处理、净化。
Amberlite IRN77 is a strong acid cation exchange resin.

Application

Amberlite IRN77 was used as cation exchanger to determine the sorption activity towards Cs(I) and Sr(II) using isothermal titration calorimetry and solution-depletion methods.

Legal Information

Amberlite is a trademark of DuPont de Nemours, Inc.

pictograms

Exclamation mark

signalword

Warning

hcodes

Hazard Classifications

Eye Irrit. 2

存储类别

10 - Combustible liquids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves, type ABEK (EN14387) respirator filter

法规信息

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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Kyeong-Ho Yeon et al.
Water research, 38(7), 1911-1921 (2004-03-18)
This study investigated the production of high-purity water in the primary coolant of a nuclear power plant via the continuous electrodeionization (CEDI) process, using ion exchange resins as ion-conducting media between ion exchange membranes. The effectiveness of this method was
Benedicte Prelot et al.
Environmental science and pollution research international, 21(15), 9334-9343 (2014-04-15)
Sorption performance of cation-exchange resins Amberlite® IRN77 and Amberlite™ IRN9652 toward Cs(I) and Sr(II) has been tested in single-component aqueous solutions and simulated waste effluents containing other monovalent (Effluent 1) or divalent (Effluent 2) metal cations, as well as nitrate
Cristina Morales Torres et al.
Nature communications, 11(1), 1792-1792 (2020-04-15)
Continuous cancer growth is driven by subsets of self-renewing malignant cells. Targeting of uncontrolled self-renewal through inhibition of stem cell-related signaling pathways has proven challenging. Here, we show that cancer cells can be selectively deprived of self-renewal ability by interfering

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