InChI
1S/C20H12O5/c21-11-5-7-15-17(9-11)24-18-10-12(22)6-8-16(18)20(15)14-4-2-1-3-13(14)19(23)25-20/h1-10,21-22H
InChI key
GNBHRKFJIUUOQI-UHFFFAOYSA-N
SMILES string
Oc1ccc2c(Oc3cc(O)ccc3C24OC(=O)c5ccccc45)c1
mp
320 °C (lit.)
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signalword
Warning
hcodes
pcodes
Hazard Classifications
Eye Irrit. 2
存储类别
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
新产品
此项目有
Manuel Sánchez-de-la-Torre et al.
Journal of the American College of Cardiology, 66(9), 1023-1032 (2015-09-01)
In patients with resistant hypertension (RH) and obstructive sleep apnea (OSA), the blood pressure response to continuous positive airway pressure (CPAP) treatment is highly variable and could be associated with differential micro-ribonucleic acid (miRNA) profiles. Currently, no available methods exist
René C L Olsthoorn
Nucleic acids research, 42(14), 9327-9333 (2014-07-18)
Cellular ribonucleic acid (RNA) plays a crucial role in the initial conversion of cellular prion protein PrP(C) to infectious PrP(Sc) or scrapie. The nature of this RNA remains elusive. Previously, RNA aptamers against PrP(C) have been isolated and found to
Jan Fiedler et al.
Journal of the American College of Cardiology, 66(18), 2005-2015 (2015-10-31)
Long noncoding ribonucleic acids (lncRNAs) are a subclass of regulatory noncoding ribonucleic acids for which expression and function in human endothelial cells and angiogenic processes is not well studied. The authors discovered hypoxia-sensitive human lncRNAs via next-generation ribonucleic acid sequencing
Marco V Perez et al.
Journal of the American College of Cardiology, 66(22), 2522-2533 (2015-12-15)
The genetic determinants of heart failure (HF) and response to medical therapy remain unknown. We hypothesized that identifying genetic variants of HF that associate with response to medical therapy would elucidate the genetic basis of cardiac function. This study sought
Yoshiyuki Kataoka et al.
Journal of the American College of Cardiology, 63(24), 2722-2733 (2014-04-29)
This study examined the impact of omentin on myocardial injury in a mouse model of ischemia/reperfusion (I/R) and explored its underlying mechanisms. Obesity is a major risk factor for ischemic heart disease. Omentin is a circulating adipokine that is down-regulated
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