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Merck
CN

V001788

铬酸钾

puriss., 99.99%

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关于此项目

线性分子式:
K2CrO4
化学文摘社编号:
分子量:
194.19
EC Number:
232-140-5
PubChem Substance ID:
MDL number:
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vapor density

6.7 (vs air)

grade

puriss.

assay

99.99%

mp

971 °C (lit.)

SMILES string

[K+].[K+].[O-][Cr]([O-])(=O)=O

InChI

1S/Cr.2K.4O/q;2*+1;;;2*-1

InChI key

XMXNVYPJWBTAHN-UHFFFAOYSA-N

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signalword

Danger

Hazard Classifications

Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1B Inhalation - Eye Irrit. 2 - Muta. 1B - Skin Irrit. 2 - Skin Sens. 1 - STOT SE 3

target_organs

Respiratory system

存储类别

6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

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Jiwoo Hong et al.
Scientific reports, 5, 10685-10685 (2015-06-03)
We here develop a three-dimensional DMF (3D DMF) platform with patterned electrodes submerged in an oil medium to provide fundamental solutions to the technical limitations of 2D DMF platforms and water-air systems. 3D droplet manipulation on patterned electrodes is demonstrated
Aminur Rahman et al.
Journal of environmental science and health. Part A, Toxic/hazardous substances & environmental engineering, 50(11), 1136-1147 (2015-07-21)
Chromium and chromium containing compounds are discharged into the nature as waste from anthropogenic activities, such as industries, agriculture, forest farming, mining and metallurgy. Continued disposal of these compounds to the environment leads to development of various lethal diseases in
Bo Xia et al.
Toxicology letters, 228(3), 241-247 (2014-05-27)
Chromium is a potent human mutagen and carcinogen. The capability of chromium to cause cancers has been known for more than a century, and numerous epidemiological studies have been performed to determine its carcinogenicity. In the post-genome era, cancer has
Zachary DeLoughery et al.
Toxicological sciences : an official journal of the Society of Toxicology, 143(1), 54-63 (2014-10-08)
Hexavalent chromium is a human respiratory carcinogen that undergoes intracellular activation in vivo primarily via reduction with ascorbate. Replication of Cr-adducted DNA triggers mismatch repair that generates toxic DNA double-strand breaks (DSBs) as secondary lesions. Here, we examined the intranuclear distribution
Yanshuang Song et al.
Toxicology letters, 226(2), 198-205 (2014-02-25)
Our previous epidemiological study revealed that co-exposure of [Cr(VI)] with small amount of heavy metals could induce peripheral blood element imbalance, but little was known about the contribution of Cr(VI) itself and the multi-element distribution profile in other target tissues.

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