InChI
1S/C4H8O/c1-3-4(2)5/h3H2,1-2H3
InChI key
ZWEHNKRNPOVVGH-UHFFFAOYSA-N
SMILES string
CC(CC)=O
grade
ACS reagent, puriss.
agency
reag. Ph. Eur.
vapor density
2.49 (vs air)
vapor pressure
71 mmHg ( 20 °C)
assay
99.5%
autoignition temp.
960 °F
expl. lim.
10.1 %
refractive index
n20/D 1.379 (lit.)
bp
80 °C (lit.)
mp
−87 °C (lit.)
density
0.805 g/mL at 25 °C (lit.)
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signalword
Danger
hcodes
Hazard Classifications
Eye Irrit. 2 - Flam. Liq. 2 - STOT SE 3
target_organs
Central nervous system
supp_hazards
存储类别
3 - Flammable liquids
wgk
WGK 1
flash_point_f
30.2 °F - closed cup
flash_point_c
-1 °C - closed cup
法规信息
新产品
此项目有
Jeong-Hwan Yoon et al.
Nature communications, 6, 7600-7600 (2015-07-22)
Transforming growth factor-β (TGF-β) and interleukin-6 (IL-6) are the pivotal cytokines to induce IL-17-producing CD4(+) T helper cells (TH17); yet their signalling network remains largely unknown. Here we show that the highly homologous TGF-β receptor-regulated Smads (R-Smads): Smad2 and Smad3
Sherille D Bradley et al.
Cancer immunology research, 3(6), 602-609 (2015-03-22)
Oncogene activation in tumor cells induces broad and complex cellular changes that contribute significantly to disease initiation and progression. In melanoma, oncogenic BRAF(V600E) has been shown to drive the transcription of a specific gene signature that can promote multiple mechanisms
Federico Mele et al.
Nature communications, 6, 6431-6431 (2015-03-17)
T helper (TH) cell polarization during priming is modulated by a number of signals, but whether polarization to a given phenotype also influences recall responses of memory TH cells is relatively unknown. Here we show that miR-181a is selectively induced
L Schafranek et al.
Leukemia, 29(1), 76-85 (2014-05-13)
Kinase inhibitors block proliferative signals in BCR-ABL1+ leukemic cells, but their capacity to induce apoptosis is poorly understood. Initial studies suggested that very brief exposure to kinase inhibitors was sufficient to induce apoptosis in chronic myeloid leukemia (CML) cells. However
S I Labidi-Galy et al.
Oncogene, 34(3), 373-383 (2014-01-29)
High-grade serous ovarian carcinoma (HGSOC) and basal-like breast cancer (BLBC) share many features including TP53 mutations, genomic instability and poor prognosis. We recently reported that Elafin is overexpressed by HGSOC and is associated with poor overall survival. Here, we confirm
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