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Merck
CN

V003437

白蛋白 来源于鸡蛋白

别名:

卵清蛋白, 清蛋白 来源于鸡蛋白

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化学文摘社编号:
EC Number:
232-692-7
MDL number:
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mol wt

44287 Da by calculation

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Application

磷酸化糖蛋白,可用作蛋白质电泳的标记物(~45kDa)。已被用于生产哮喘的动物模型。

pictograms

Health hazard

signalword

Danger

hcodes

Hazard Classifications

Resp. Sens. 1A

存储类别

11 - Combustible Solids

wgk

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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JangEun Lee et al.
Journal of immunology (Baltimore, Md. : 1950), 192(7), 3190-3199 (2014-02-26)
Fatty acids (FAs) are essential constituents of cell membranes, signaling molecules, and bioenergetic substrates. Because CD8(+) T cells undergo both functional and metabolic changes during activation and differentiation, dynamic changes in FA metabolism also occur. However, the contributions of de
G H Grit et al.
Parasite immunology, 36(4), 157-169 (2014-01-01)
Giardia duodenalis is an important intestinal parasite in animals and humans. The role of dendritic cells (DC) in the initiation of the immune response against G. duodenalis is poorly documented. The aim of this study was to test the hypothesis that
Rylie P Flesher et al.
Clinical science (London, England : 1979), 126(11), 805-814 (2013-12-19)
Endogenous mediators, such as RvE1 (resolvin E1), promote resolution of an inflammatory response and have potential as novel therapeutic agents. In the present study, we investigated the activity of RvE1 in a model of an acute exacerbation of chronic allergic
Sheng Zeng et al.
The Journal of asthma : official journal of the Association for the Care of Asthma, 51(4), 360-365 (2014-02-26)
It has been reported that adoptive transfer of γδ T cells increases the cellular infiltration, especially eosinophils, in the lungs of allergic mice, suggesting that γδ T cells may play a proinflammatory role in allergic airway inflammation. Respiratory syncytial virus
Natalia Korneychuk et al.
Gastroenterology, 146(4), 1017-1027 (2013-12-24)
CD4(+) T cells specific for dietary gluten and interleukin 15 (IL15) contribute to the pathogenesis of celiac disease. We investigated whether and how they interact to damage the intestine using mice that overexpress human IL15 in the intestinal epithelium and

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