V800247
氯化锰
AR, ≥99%
别名:
二氯化锰, 黄钙橄石
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线性分子式:
MnCl2
化学文摘社编号:
分子量:
125.84
UNSPSC Code:
12352302
PubChem Substance ID:
MDL number:
eCl@ss:
38190105
Assay:
≥99%
Grade:
AR
Form:
crystalline powder
InChI
1S/2ClH.Mn/h2*1H;/q;;+2/p-2
SMILES string
Cl[Mn]Cl
InChI key
GLFNIEUTAYBVOC-UHFFFAOYSA-L
grade
AR
product line
Vetec™
assay
≥99%
form
crystalline powder
pH
5.0-6.0 (5%)
mp
652 °C (lit.)
density
2.98 g/mL at 25 °C (lit.)
anion traces
sulfate (SO42-): ≤0.005%
cation traces
Ba: ≤0.01%, Ca: ≤0.03%, Cu: ≤0.0005%, Fe: ≤0.0005%, K: ≤0.01%, Mg: ≤0.005%, Na: ≤0.05%, Ni: ≤0.002%, Zn: ≤0.005%, heavy metals (as Pb): ≤0.0005%
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Legal Information
Vetec is a trademark of Merck KGaA, Darmstadt, Germany
signalword
Danger
hcodes
Hazard Classifications
Acute Tox. 3 Oral - Eye Dam. 1 - STOT RE 2
target_organs
Brain
存储类别
6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects
wgk
WGK 2
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
新产品
此项目有
Jerome A Roth et al.
Toxicology in vitro : an international journal published in association with BIBRA, 26(7), 1143-1149 (2012-07-31)
Mutations in the parkin gene are linked to development of juvenile onset of Parkinson's disease and recent studies have reported that parkin can protect against increased oxidative stress and mitochondrial dysfunction caused by a variety of oxidative and toxic insults.
A G Kanthasamy et al.
Toxicology letters, 214(3), 288-295 (2012-09-22)
The role of normal cellular prion protein (PrP) remains to be fully elucidated; however, the protein is crucial for the infection and progression of prion diseases. Recent evidence indicates that PrP is a metalloprotein since the octapeptide repeat sequences in
Jerome A Roth et al.
Neurotoxicology, 35, 121-128 (2013-01-15)
Chronic exposure to Mn results in the development of a neurological disorder known as manganism characterized by neurological deficits resembling that seen in Parkinsonism. Although dopaminergic neurons within the nigrostriatal pathway appear intact, Mn-induced irregularities in DA transmission have been
Marta Sidoryk-Wegrzynowicz et al.
Journal of neurochemistry, 122(4), 856-867 (2012-06-20)
Manganese (Mn) has been implicated in the impairment of the glutamate-glutamine cycling (GGC) by deregulation of Glu and glutamine (Gln) turnover in astrocytes. Here, we have examined possible mechanisms involved in the Mn(II)-mediated disruption of Glu turnover, including those related
Anne Bertrand et al.
NeuroImage, 64, 693-702 (2012-09-11)
The impairment of axonal transport by overexpression or hyperphosphorylation of tau is well documented for in vitro conditions; however, only a few studies on this phenomenon have been conducted in vivo, using invasive procedures, and with contradictory results. Here we
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